INFRA R therapy by R. RAKHIMOV's method
The developed method is based on normalizing metabolic processes and eliminating pathological conditions. In other words, the applied radiation normalizes metabolic processes and addresses the root cause of the disease rather than just its symptoms.
Ceramic infrared (IR) emitters used in medicine have a specific wavelength within a narrow spectral range. Moreover, they have various temporal characteristics and can operate in continuous, pulsed modes, or emit energy in a complex temporal sequence. Structurally, there are emitters for general and localized applications. General emitters affect the entire body surface, while localized emitters act on specific organs or body areas involved in the pathological process.
Ceramic infrared (IR) emitters used in medicine have a specific wavelength within a narrow spectral range. Moreover, they have various temporal characteristics and can operate in continuous, pulsed modes, or emit energy in a complex temporal sequence. Structurally, there are emitters for general and localized applications. General emitters affect the entire body surface, while localized emitters act on specific organs or body areas involved in the pathological process.
CHARACTERISTICS OF IR EMITTERS AND THEIR MECHANISM OF ACTION
All types of emitters are characterized by the fact that their energy spectrum (E = hν) corresponds to or is lower than the energy spectrum of human radiation. In each specific case, the emission range must be energetically (spectrally) in resonance with the process that needs to be influenced. Therefore, emitters have an effect only when there is a pathological focus in the body (its absorption spectrum differs from that of a healthy person). Emitters in these ranges do not affect a healthy person in any way.
Emitters of the K series have a spectrum that is identical or very close to the range of natural human radiation.
The peak of human radiation is at 9.36 μm. It should be noted that with age, this peak gradually shifts to a longer wavelength region. Whether this is due to DNA reduction or disruptions in microcirculation and the drainage system is difficult to determine precisely. Most likely, both factors influence the condition of the body. Observations of twins indicate that lifestyle affects their physiological state.
K series emitters, by harmonizing the rates of chemical and biochemical processes, exert an immunomodulatory and stimulating effect on the body. Additionally, they can normalize hypothalamic function, stimulate endocrine glands, and regulate pancreatic function.
GI and AF emitters act on human pathogenic flora, exhibiting antimicrobial and anti-inflammatory properties while also being lethal to protozoa.
R series emitters facilitate recombination of radicals with high activation energy and have anti-oncological and antiviral effects.
K Series Emitters
In the human body, various processes continuously take place, forming a chain of chemical reactions occurring in a strict sequence. When exposed to pathogenic factors, one or more of these processes may be disrupted, leading to an increase or decrease in the rate of one or more chemical reactions. As a result, there is either a deficiency or an excess of components that should have been consumed in these reactions and converted into other compounds necessary for subsequent processes. This disruption leads to a failure of the entire well-regulated chain, making it impossible to complete the full normal metabolic cycle. Clinically, this manifests as various diseases (such as foreign tissue deposition and stone formation in different organs), vascular dysfunction, endocrine disorders, allergic reactions, skin diseases, reduced immunity, and more. A sick person lacks an adequate response to the impact of various pathogenic agents.
It is important to remember that most metabolic processes in the human body are photochemical and resonate within the range of the body’s natural radiation. Therefore, the rate and coordination of these processes strictly depend on the intensity of this radiation. Excessive radiation does not have a negative impact since reaction rates do not increase indefinitely; they are limited by the availability of the necessary components for a particular reaction at a given moment. Additionally, the rate of chemical reactions can be regulated by the nervous system.
It is well known that although a healthy young and elderly person have the same body temperature, they differ in the intensity (flux) of emitted infrared (IR) radiation. The level of this radiation indicates the body's vitality; accordingly, the activity of biochemical processes in younger individuals is higher and more synchronized than in older ones. A reduced IR radiation level is also observed in individuals with weakened immune systems, critically ill patients, and those near death.
K series IR emitters mimic human radiation at its characteristic wavelength, positively affecting metabolism and immunity by stimulating and activating the body's natural processes. When using a general-purpose emitter, the body receives the necessary infrared radiation at its natural wavelength, restoring the chain and speed of ongoing biochemical processes. Once these processes return to normal, the body's own IR radiation production is restored, reinforcing the normalization of chemical reaction rates and metabolic processes. As a result, patients experience improved immunity, homeostasis, organ function, circulation, overall well-being, reduced fatigue, normalized sleep, and appetite.
Emitters of the K series have a spectrum that is identical or very close to the range of natural human radiation.
The peak of human radiation is at 9.36 μm. It should be noted that with age, this peak gradually shifts to a longer wavelength region. Whether this is due to DNA reduction or disruptions in microcirculation and the drainage system is difficult to determine precisely. Most likely, both factors influence the condition of the body. Observations of twins indicate that lifestyle affects their physiological state.
K series emitters, by harmonizing the rates of chemical and biochemical processes, exert an immunomodulatory and stimulating effect on the body. Additionally, they can normalize hypothalamic function, stimulate endocrine glands, and regulate pancreatic function.
GI and AF emitters act on human pathogenic flora, exhibiting antimicrobial and anti-inflammatory properties while also being lethal to protozoa.
- AF emitters also have antifungal effects.
- GI emitters help normalize intestinal microflora, activate macrophages, convert hormones into their active state, and more.
R series emitters facilitate recombination of radicals with high activation energy and have anti-oncological and antiviral effects.
Z series emitters act on pathological tissues and improve microcirculation.
K Series Emitters
In the human body, various processes continuously take place, forming a chain of chemical reactions occurring in a strict sequence. When exposed to pathogenic factors, one or more of these processes may be disrupted, leading to an increase or decrease in the rate of one or more chemical reactions. As a result, there is either a deficiency or an excess of components that should have been consumed in these reactions and converted into other compounds necessary for subsequent processes. This disruption leads to a failure of the entire well-regulated chain, making it impossible to complete the full normal metabolic cycle. Clinically, this manifests as various diseases (such as foreign tissue deposition and stone formation in different organs), vascular dysfunction, endocrine disorders, allergic reactions, skin diseases, reduced immunity, and more. A sick person lacks an adequate response to the impact of various pathogenic agents.
It is important to remember that most metabolic processes in the human body are photochemical and resonate within the range of the body’s natural radiation. Therefore, the rate and coordination of these processes strictly depend on the intensity of this radiation. Excessive radiation does not have a negative impact since reaction rates do not increase indefinitely; they are limited by the availability of the necessary components for a particular reaction at a given moment. Additionally, the rate of chemical reactions can be regulated by the nervous system.
It is well known that although a healthy young and elderly person have the same body temperature, they differ in the intensity (flux) of emitted infrared (IR) radiation. The level of this radiation indicates the body's vitality; accordingly, the activity of biochemical processes in younger individuals is higher and more synchronized than in older ones. A reduced IR radiation level is also observed in individuals with weakened immune systems, critically ill patients, and those near death.
K series IR emitters mimic human radiation at its characteristic wavelength, positively affecting metabolism and immunity by stimulating and activating the body's natural processes. When using a general-purpose emitter, the body receives the necessary infrared radiation at its natural wavelength, restoring the chain and speed of ongoing biochemical processes. Once these processes return to normal, the body's own IR radiation production is restored, reinforcing the normalization of chemical reaction rates and metabolic processes. As a result, patients experience improved immunity, homeostasis, organ function, circulation, overall well-being, reduced fatigue, normalized sleep, and appetite.
Based on the emission spectrum, K series emitters are classified into:
Local emitters of the K series affect zones sensitive to perception. These areas include the projection of the heels, thymus, pancreas, the VII cervical vertebra, and the hypothalamus. By directly influencing these zones, it is possible to enhance the body's adaptive capabilities and its resistance.
In practice, the KS emitter is rarely used in patient treatment, as they often have weakened immunity and a reduced overall metabolic rate. Considering that both the body and pathogenic microorganisms share a common source of nutrients, it is advisable to accelerate the body's consumption of plastic and energy materials. This way, the restorative processes in the body will outpace the development of infection, leading to faster recovery. Therefore, it is most appropriate to use the KL and KB emitters.
The KH emitter is used only as "emergency aid" to restore the balance between the sympathetic and parasympathetic nervous systems. According to our classification, the sympathetic system is responsible for action, while the parasympathetic system protects organs, tissues, and body systems from the negative effects of reactions stimulated by the sympathetic system.
The primary emitter is KL, as it stimulates the sympathetic system, harmonizes metabolic processes in the body, including the balance between the sympathetic and parasympathetic systems, and accelerates restorative processes. It also alleviates allergic and hyperreactive responses by normalizing the speed of bodily processes. This promotes the elimination of substances not involved in normal metabolism.
Let us examine in more detail its effects on immunoactive zones:
The R series emitters
According to modern scientific understanding, free radicals play a significant role in the development of cancer, cardiovascular diseases, and other conditions at the chemical level.
A free radical is an atom or molecule with an unpaired electron in its outer orbit. This makes it a highly chemically active element, as it seeks to either gain or lose an electron to form a stable electron pair. Molecules that typically have double bonds, through which this process occurs, become reactive themselves, as the unpaired electron remains unpaired. The presence of such radicals is the starting point of a chain reaction. The absence of a biological compound (or another factor) capable of quickly eliminating free radicals, or the presence of radicals with high activation energy for recombination, leads to irreversible cell damage.
Under normal conditions, free radicals are produced during certain physiological processes. After completing their functions, radicals recombine, forming stable molecules or compounds. However, their abnormal production can occur in many cases. For example, it may result from disruptions in electron transport within the mitochondrial respiratory chain, which is often observed in the metabolism of cancer cells. Additionally, free radicals play a key role in the active proliferation of cancer cells. The destructive role of excess free radicals has been proven in the development of many diseases, including gastrointestinal disorders, pancreatitis, diabetes, myocardial infarction, and others. The formation of radicals can be triggered by viruses, radiation, UV exposure, pulsed electromagnetic fields, chemical toxins (such as dioxin), impaired blood circulation, heavy metal ions, excess oxygen, or the presence of strong oxidizers (e.g., ozone), among other factors.
The elimination of free radicals is actively supported by the body's antioxidant defense mechanisms, both inside and outside the cell. However, in the case of degeneratively altered cancer cells, these antioxidant defenses fail.
Firstly, the number of free radicals continuously increases.
Secondly, in cancer cells, the steric factor and the charge of the active part (the enzyme-substrate complex), responsible for cell growth and radical recombination after completing their functions, are altered. This, in turn, changes the energy barrier for the recombination reaction. In chemical terms, the process has a high activation energy or energy barrier specifically for recombination, which should conclude the chain growth process. This also makes it difficult or impossible for antioxidants to eliminate oncological radicals. The activation energy for the recombination of such radicals is significantly higher than normal, so the body's existing defense mechanisms cannot inactivate them. This does not mean that the activation energy for side reactions also increases. On the contrary, conditions are likely created for their occurrence. As a result, the growth of cancer cells becomes uncontrolled, and external intervention is necessary to actively participate in the elimination of oncological radicals.
Let us consider the mechanism of cell synthesis using DNA replication as an example.
There is a key enzyme E, such as telomerase, which carries out DNA synthesis. It is important to note that enzymes are highly specific. In other words, only reactions with a specific substrate have a low energy barrier. In reality, enzymes facilitate chemical reactions by lowering the activation energy of a specific reaction. If there are several substrates S1, S2, S3, etc., this means that the enzyme-substrate complex forms only with the substrate whose chemical reaction has an energy barrier that can be overcome under given conditions. For example, let this be S1:
The attachment of S1 to the enzyme changes enzyme E into ES1. This is now a different enzyme, and it activates another reaction, for example, with S2:
The superoxide radical, due to its high reactivity, can attach to the enzyme, overcoming even a high energy barrier at any stage of DNA growth. In this case, the altered main enzyme will synthesize completely different molecules. As a result, atypical cells are formed. In our opinion, this is the primary mechanism of their formation.
The RC emitter is a universal eliminator of free radicals. Its mechanism of action lies in its ability to overcome the activation energy (energy barrier), thereby increasing the reactivity of growing "foreign" radicals and facilitating their recombination with an auxiliary radical. Let us explain this in more detail.
Free radicals, as highly reactive molecules with unpaired electrons, seek to stabilize by gaining or losing electrons. However, in pathological conditions, such as cancer, the energy barrier for recombination of these radicals becomes too high, making it difficult for the body's natural antioxidant systems to neutralize them. This leads to uncontrolled chain reactions and cellular damage.
The RC emitter works by providing the necessary energy to overcome this barrier. It enhances the reactivity of the radicals, making them more likely to recombine with other radicals or molecules, thereby neutralizing their harmful effects. Essentially, the emitter acts as a catalyst, lowering the energy threshold required for recombination and enabling the body's natural processes to eliminate these radicals more effectively.
In simpler terms, the RC emitter helps "push" the radicals over the energy barrier, allowing them to recombine and stabilize, rather than continuing to cause damage. This process is particularly crucial in conditions like cancer, where the radicals involved have abnormally high activation energies, making them resistant to the body's usual defense mechanisms. By facilitating recombination, the RC emitter helps restore balance and prevent further cellular damage.
In practice, the KS emitter is rarely used in patient treatment, as they often have weakened immunity and a reduced overall metabolic rate. Considering that both the body and pathogenic microorganisms share a common source of nutrients, it is advisable to accelerate the body's consumption of plastic and energy materials. This way, the restorative processes in the body will outpace the development of infection, leading to faster recovery. Therefore, it is most appropriate to use the KL and KB emitters.
The KH emitter is used only as "emergency aid" to restore the balance between the sympathetic and parasympathetic nervous systems. According to our classification, the sympathetic system is responsible for action, while the parasympathetic system protects organs, tissues, and body systems from the negative effects of reactions stimulated by the sympathetic system.
The primary emitter is KL, as it stimulates the sympathetic system, harmonizes metabolic processes in the body, including the balance between the sympathetic and parasympathetic systems, and accelerates restorative processes. It also alleviates allergic and hyperreactive responses by normalizing the speed of bodily processes. This promotes the elimination of substances not involved in normal metabolism.
Let us examine in more detail its effects on immunoactive zones:
- Heel bones: When exposed to this radiation, increased blood circulation is observed.
- Thymus: The thymus is one of the main regulators of bodily processes. It is now known to produce over 20 active substances that help maintain high immunity. Additionally, it synthesizes T-lymphocytes, which are the primary "recognizers" of pathological factors. With age, the thymus shrinks. In our view, this is due to the fact that during periods of rapid growth in a young organism, significant shifts occur in all systems. The thymus, due to its broad range of action, helps maintain high immunity and the normal state of the body. Once growth is complete, such global processes are no longer observed, and the need for thymus metabolites decreases, ultimately leading to a reduction in its size. Children with low immunity often have an enlarged thymus.
The R series emitters
According to modern scientific understanding, free radicals play a significant role in the development of cancer, cardiovascular diseases, and other conditions at the chemical level.
A free radical is an atom or molecule with an unpaired electron in its outer orbit. This makes it a highly chemically active element, as it seeks to either gain or lose an electron to form a stable electron pair. Molecules that typically have double bonds, through which this process occurs, become reactive themselves, as the unpaired electron remains unpaired. The presence of such radicals is the starting point of a chain reaction. The absence of a biological compound (or another factor) capable of quickly eliminating free radicals, or the presence of radicals with high activation energy for recombination, leads to irreversible cell damage.
Under normal conditions, free radicals are produced during certain physiological processes. After completing their functions, radicals recombine, forming stable molecules or compounds. However, their abnormal production can occur in many cases. For example, it may result from disruptions in electron transport within the mitochondrial respiratory chain, which is often observed in the metabolism of cancer cells. Additionally, free radicals play a key role in the active proliferation of cancer cells. The destructive role of excess free radicals has been proven in the development of many diseases, including gastrointestinal disorders, pancreatitis, diabetes, myocardial infarction, and others. The formation of radicals can be triggered by viruses, radiation, UV exposure, pulsed electromagnetic fields, chemical toxins (such as dioxin), impaired blood circulation, heavy metal ions, excess oxygen, or the presence of strong oxidizers (e.g., ozone), among other factors.
The elimination of free radicals is actively supported by the body's antioxidant defense mechanisms, both inside and outside the cell. However, in the case of degeneratively altered cancer cells, these antioxidant defenses fail.
Firstly, the number of free radicals continuously increases.
Secondly, in cancer cells, the steric factor and the charge of the active part (the enzyme-substrate complex), responsible for cell growth and radical recombination after completing their functions, are altered. This, in turn, changes the energy barrier for the recombination reaction. In chemical terms, the process has a high activation energy or energy barrier specifically for recombination, which should conclude the chain growth process. This also makes it difficult or impossible for antioxidants to eliminate oncological radicals. The activation energy for the recombination of such radicals is significantly higher than normal, so the body's existing defense mechanisms cannot inactivate them. This does not mean that the activation energy for side reactions also increases. On the contrary, conditions are likely created for their occurrence. As a result, the growth of cancer cells becomes uncontrolled, and external intervention is necessary to actively participate in the elimination of oncological radicals.
Let us consider the mechanism of cell synthesis using DNA replication as an example.
There is a key enzyme E, such as telomerase, which carries out DNA synthesis. It is important to note that enzymes are highly specific. In other words, only reactions with a specific substrate have a low energy barrier. In reality, enzymes facilitate chemical reactions by lowering the activation energy of a specific reaction. If there are several substrates S1, S2, S3, etc., this means that the enzyme-substrate complex forms only with the substrate whose chemical reaction has an energy barrier that can be overcome under given conditions. For example, let this be S1:
E + S1 = ES1
During the growth process, the enzyme-substrate complex represents a radical.The attachment of S1 to the enzyme changes enzyme E into ES1. This is now a different enzyme, and it activates another reaction, for example, with S2:
ES1 + S2 = ES1S2
If the activation energy of the newly formed enzyme ES1S2 for substrate S1 decreases, then:ES1S2 + S1 = ES1S2S1
As follows from this mechanism, a small number of substrates can produce an infinite variety of molecules, such as DNA or RNA.The superoxide radical, due to its high reactivity, can attach to the enzyme, overcoming even a high energy barrier at any stage of DNA growth. In this case, the altered main enzyme will synthesize completely different molecules. As a result, atypical cells are formed. In our opinion, this is the primary mechanism of their formation.
The RC emitter is a universal eliminator of free radicals. Its mechanism of action lies in its ability to overcome the activation energy (energy barrier), thereby increasing the reactivity of growing "foreign" radicals and facilitating their recombination with an auxiliary radical. Let us explain this in more detail.
Free radicals, as highly reactive molecules with unpaired electrons, seek to stabilize by gaining or losing electrons. However, in pathological conditions, such as cancer, the energy barrier for recombination of these radicals becomes too high, making it difficult for the body's natural antioxidant systems to neutralize them. This leads to uncontrolled chain reactions and cellular damage.
The RC emitter works by providing the necessary energy to overcome this barrier. It enhances the reactivity of the radicals, making them more likely to recombine with other radicals or molecules, thereby neutralizing their harmful effects. Essentially, the emitter acts as a catalyst, lowering the energy threshold required for recombination and enabling the body's natural processes to eliminate these radicals more effectively.
In simpler terms, the RC emitter helps "push" the radicals over the energy barrier, allowing them to recombine and stabilize, rather than continuing to cause damage. This process is particularly crucial in conditions like cancer, where the radicals involved have abnormally high activation energies, making them resistant to the body's usual defense mechanisms. By facilitating recombination, the RC emitter helps restore balance and prevent further cellular damage.
Fig. 1. Temporal characteristics of the RC emitter. P – pulse energy density.
The radiation produced by this ceramic consists of two powerful short pulses with an intensity of up to 320 W/cm² and a duration of 10-12 microseconds each, following one after the other. Each pulse has its own optical range (16 and 16.25 µm) and, accordingly, quantum energy. The first pulse leads to the formation of short-lived radicals from water or lipid molecules, while the second pulse causes them to resonantly recombine with oncological radicals and among themselves.
The wavelength in the 16 µm range was chosen from the perspective of radiation safety. This energy does not have a damaging effect on our normal molecules and tissues. The decisive role in activating specific processes is played not only by the spectral composition of the radiation generated by the functional ceramic but also by the rate of pulse rise.
Research conducted by us in collaboration with Professor Choi showed that the level of free radicals in cancer patients is 5-6 times higher than normal. After exposure to the RC emitter, their levels normalized.
A virus consists of DNA or RNA with a corresponding enzyme that copies it. For example, hepatitis A has RNA polymerase, hepatitis B has DNA polymerase, and the enzyme of the AIDS virus is reverse transcriptase, and so on.
At the moment of virus replication (like any living organism), an enzyme-substrate complex is formed, which represents a growing radical. The pulsed radiation generated by the functional ceramic of the R series creates auxiliary radicals from water or lipid molecules (first pulse). The second pulse recombines the enzyme-substrate complex of the virus (the growing radical) with this auxiliary radical. As a result, the enzyme is inactivated and cannot synthesize viruses.
The RV emitter differs from the RC in that it generates two additional pulses at 8 and 8.125 µm and also has antiviral and anticancer effects by inactivating the corresponding enzymes. The generation of additional pulses is achieved by replacing part of the lanthanum oxide with neodymium oxide.
Thus, this type of radiation acts on radicals with special properties in damaged tissue, which drive the pathological process.
The wavelength in the 16 µm range was chosen from the perspective of radiation safety. This energy does not have a damaging effect on our normal molecules and tissues. The decisive role in activating specific processes is played not only by the spectral composition of the radiation generated by the functional ceramic but also by the rate of pulse rise.
Research conducted by us in collaboration with Professor Choi showed that the level of free radicals in cancer patients is 5-6 times higher than normal. After exposure to the RC emitter, their levels normalized.
A virus consists of DNA or RNA with a corresponding enzyme that copies it. For example, hepatitis A has RNA polymerase, hepatitis B has DNA polymerase, and the enzyme of the AIDS virus is reverse transcriptase, and so on.
At the moment of virus replication (like any living organism), an enzyme-substrate complex is formed, which represents a growing radical. The pulsed radiation generated by the functional ceramic of the R series creates auxiliary radicals from water or lipid molecules (first pulse). The second pulse recombines the enzyme-substrate complex of the virus (the growing radical) with this auxiliary radical. As a result, the enzyme is inactivated and cannot synthesize viruses.
The RV emitter differs from the RC in that it generates two additional pulses at 8 and 8.125 µm and also has antiviral and anticancer effects by inactivating the corresponding enzymes. The generation of additional pulses is achieved by replacing part of the lanthanum oxide with neodymium oxide.
Thus, this type of radiation acts on radicals with special properties in damaged tissue, which drive the pathological process.
G Series Emitters
The action of this emitter is based on the principle of "low-temperature" sterilization. Its mechanism involves interrupting certain processes (including chemical reactions) necessary for cell reproduction. All microorganisms reproduce through division. At the molecular level, this process represents a chain of chemical reactions in a strict sequence, which ultimately leads to the process of reproduction.
The principle of constructing such a chain can be schematically represented as a system:
The GI emitter has an anti-inflammatory effect, the mechanism of which is based on the destruction of pathogenic flora, stimulation of the development of normal microflora, reduction of vascular wall permeability, and an increase in the level of free steroids in the blood due to the dissociation of the protein-steroid complex. It also activates the adrenal glands, dissociates lipid-cholesterol complexes, releases free cholesterol, which is used for the synthesis of corticosteroids, and stimulates macrophages. Studies have shown that a 10-minute exposure to this emitter stimulates macrophages, and their activity persists for at least a month.
The emitter is highly effective in dissolving excess fatty tissue, including lipomas.
The action of this emitter is based on the principle of "low-temperature" sterilization. Its mechanism involves interrupting certain processes (including chemical reactions) necessary for cell reproduction. All microorganisms reproduce through division. At the molecular level, this process represents a chain of chemical reactions in a strict sequence, which ultimately leads to the process of reproduction.
The principle of constructing such a chain can be schematically represented as a system:
a b —- a + b
c d —- c + d
a c —- a + c
b d —- b +d
……………………..
When exposed to the GI emitter, certain chemical reactions in the above-described chain are interrupted, making it impossible to complete the process of cell division. As a result, microorganisms die because their life cycle is very short. Exposure to the radiation for 45 minutes is sufficient to destroy pathogenic microorganisms (staphylococci, streptococci, normalize the concentration of Koch's bacilli, etc.), while 1.5 hours is enough to eliminate protozoa (Trichomonas, Giardia, etc.).The GI emitter has an anti-inflammatory effect, the mechanism of which is based on the destruction of pathogenic flora, stimulation of the development of normal microflora, reduction of vascular wall permeability, and an increase in the level of free steroids in the blood due to the dissociation of the protein-steroid complex. It also activates the adrenal glands, dissociates lipid-cholesterol complexes, releases free cholesterol, which is used for the synthesis of corticosteroids, and stimulates macrophages. Studies have shown that a 10-minute exposure to this emitter stimulates macrophages, and their activity persists for at least a month.
The emitter is highly effective in dissolving excess fatty tissue, including lipomas.
Emitter AF
The mechanism of action of the AF emitter is based on the same principle as the GI emitter. However, unlike the GI emitter, the energy of AF disrupts a greater number of reactions involved in the reproduction of microorganisms. Its range of action extends not only to pathogenic bacteria and protozoa but also to pathogenic fungal flora.
The optimal exposure time for the AF emitter is shorter than that of the GI emitter (a 20-minute exposure with AF is approximately equivalent to a 60-minute exposure with GI). To eliminate pathogenic microorganisms, an exposure of just 15 minutes is sufficient.
The AF emitter can also be used for removing fluid from pathologically altered organs (such as cysts). After treatment of the intestines (for the eradication of fungal flora), the GI emitter should be used for 5-10 minutes to restore normal intestinal microflora.
The functional ceramics of both GI and AF emitters are based on RC and RV, "diluted with mullite" at a ratio of 200:1 and 25:1, respectively. Mullite, synthesized using a special technology, has a transmission spectrum of up to 25 µm. Emitters based on RV have an additional index "n" (neodymium) in their designation, corresponding to GIn and AFn.
The mechanism of action of the AF emitter is based on the same principle as the GI emitter. However, unlike the GI emitter, the energy of AF disrupts a greater number of reactions involved in the reproduction of microorganisms. Its range of action extends not only to pathogenic bacteria and protozoa but also to pathogenic fungal flora.
The optimal exposure time for the AF emitter is shorter than that of the GI emitter (a 20-minute exposure with AF is approximately equivalent to a 60-minute exposure with GI). To eliminate pathogenic microorganisms, an exposure of just 15 minutes is sufficient.
The AF emitter can also be used for removing fluid from pathologically altered organs (such as cysts). After treatment of the intestines (for the eradication of fungal flora), the GI emitter should be used for 5-10 minutes to restore normal intestinal microflora.
The functional ceramics of both GI and AF emitters are based on RC and RV, "diluted with mullite" at a ratio of 200:1 and 25:1, respectively. Mullite, synthesized using a special technology, has a transmission spectrum of up to 25 µm. Emitters based on RV have an additional index "n" (neodymium) in their designation, corresponding to GIn and AFn.
Emitter Series Z
The action of emitters in this series is based on "loosening" intermolecular bonds between molecules of foreign tissue. The chemical bonds of molecules in our body are relatively strong. However, the bonds in connective tissue formed as a result of pathological processes are weaker, as they are primarily held together by hydrogen bonds, Van der Waals forces, and other weak interactions. Under the influence of the emitters, these relatively weak bonds are broken. Pathological compounds can be converted into a soluble state, allowing their elimination from the body through blood circulation and liver function. In particular, pathological collagen is transformed into gelatin, which dissolves in the blood and is excreted by the liver.
This emitter is effective in treating angiopathy, cirrhosis, rheumatoid diseases, and more.
The action of emitters in this series is based on "loosening" intermolecular bonds between molecules of foreign tissue. The chemical bonds of molecules in our body are relatively strong. However, the bonds in connective tissue formed as a result of pathological processes are weaker, as they are primarily held together by hydrogen bonds, Van der Waals forces, and other weak interactions. Under the influence of the emitters, these relatively weak bonds are broken. Pathological compounds can be converted into a soluble state, allowing their elimination from the body through blood circulation and liver function. In particular, pathological collagen is transformed into gelatin, which dissolves in the blood and is excreted by the liver.
This emitter is effective in treating angiopathy, cirrhosis, rheumatoid diseases, and more.
INDICATIONS FOR USE OF GENERAL AND LOCAL EMITTERS
THE CONCEPT OF DISEASE AND IMMUNITY
A disease is the body's response that develops when pathogenic factors invade and is aimed at restoring homeostasis.
What causes can lead to the development of a pathological process?
In acute diseases, clinical manifestations reflect the cause of their onset. In chronic processes, however, the primary cause often becomes secondary in clinical symptoms or remains asymptomatic. The patient’s complaints are usually related more to the consequences of the initial impact. In any case, effective treatment is only achieved by eliminating the root cause of the disease.
What causes can lead to the development of a pathological process?
In acute diseases, clinical manifestations reflect the cause of their onset. In chronic processes, however, the primary cause often becomes secondary in clinical symptoms or remains asymptomatic. The patient’s complaints are usually related more to the consequences of the initial impact. In any case, effective treatment is only achieved by eliminating the root cause of the disease.
The causes of disease development may include:
- Infection (bacterial, viral, fungal).
- Body burden with various xenobiotics (heavy metal salts, toxin overload, etc.).
- Hormonal imbalance.
- Disruptions in regulatory mechanisms.
- Impaired blood circulation in the body, an organ, or its part.
- Injuries.
The predisposing factors in the development of a pathological process include:
Hereditary predisposition, including factors such as:- Type of nervous system, which determines the speed and nature of responses to various influences.
- Activity of certain metabolic processes, such as the acetylation phenotype, which affects the rate of xenobiotic inactivation.
- HLA antigen carriage, which determines susceptibility to certain diseases, etc.
- Low or high temperatures, sudden temperature changes.
- Exposure to excessive radiation, UV rays, ozone, and other oxidizers.
- Poor posture.
- Negative psycho-emotional stress.
- Harmful habits—smoking, alcohol consumption.
- Nutritional disorders, including excessive consumption of sugary and fatty foods, etc.
Today, it is well known that the defining factor in the development of a disease is the state of the body's immune system. The condition of the immune system determines the body's resistance to foreign agents. All the body's responses to external and internal stimuli are controlled by the nervous system. It precisely identifies the nature of the stimulus and determines how our metabolism should adapt to counteract, neutralize, destroy, compensate for, or eliminate the irritant (such as an infection, toxins, or disturbances caused by psycho-emotional stress).
The nervous system responds to stimuli by releasing hormones, enzymes, triggering vascular spasms, producing antibodies, and so on. If a stimulus strongly affects metabolism, the body produces more components than necessary, forcing the nervous system to neutralize the excess. When the stimulus is persistent and its intensity fluctuates chaotically, the production of active substances, their destruction, and excitatory responses occur repeatedly and unpredictably.
When the body is exposed to too many destabilizing factors—such as various infections, toxins, heavy metal ions, and especially negative psychological influences—the nervous system becomes overwhelmed. It enters a conflicting state, needing to produce active substances, destroy them immediately after, and remain ready for new production when required. This cycle—producing and releasing these active substances into the bloodstream—is essentially the immune system's response. Their destruction, once they are no longer needed, is also an immune system function.
Under such contradictory conditions, the immune system's function becomes completely disrupted. This manifests as a decrease in sympathetic nervous system activity, which can lead, for example, to reduced steroid levels or contribute to the development of autoimmune processes.
The nervous system responds to stimuli by releasing hormones, enzymes, triggering vascular spasms, producing antibodies, and so on. If a stimulus strongly affects metabolism, the body produces more components than necessary, forcing the nervous system to neutralize the excess. When the stimulus is persistent and its intensity fluctuates chaotically, the production of active substances, their destruction, and excitatory responses occur repeatedly and unpredictably.
When the body is exposed to too many destabilizing factors—such as various infections, toxins, heavy metal ions, and especially negative psychological influences—the nervous system becomes overwhelmed. It enters a conflicting state, needing to produce active substances, destroy them immediately after, and remain ready for new production when required. This cycle—producing and releasing these active substances into the bloodstream—is essentially the immune system's response. Their destruction, once they are no longer needed, is also an immune system function.
Under such contradictory conditions, the immune system's function becomes completely disrupted. This manifests as a decrease in sympathetic nervous system activity, which can lead, for example, to reduced steroid levels or contribute to the development of autoimmune processes.
From this perspective, it can be said that all processes aimed at restoring and maintaining dynamic homeostasis constitute immunity.
GUT MICROFLORA
Gut Microflora and Its Role in Health
One of the essential conditions for maintaining health is a balanced gut microflora. In all patients, regardless of the disease—whether oncological, cardiovascular, allergic, endocrine, dermatological, wound infections, mastitis, peritonitis, gangrene, and others—there is a disruption of the microflora in the small and large intestines. Often, it is impossible to restore the balance of the autonomic nervous system without normalizing intestinal function.
After chemotherapy or radiation therapy, gut microflora is completely disrupted, leading to dysfunction of the intestines. The use of the GI emitter in such cases helps restore intestinal function within 5–15 days after chemotherapy (with three daily 10-minute sessions) and 30–90 days after radiation therapy under the same regimen. Patients who have not undergone other treatments can fully restore normal intestinal function within 1–2 days.
Among patients with long-term allergic conditions, 95% recovered completely after restoring gut function. Over 50% of patients with psoriasis and other skin conditions also experienced recovery. In cases of hypertension, hypotension, and cholesterol metabolism disorders, 90% of patients recovered after intestinal function normalization and using Z-series emitters to remove pathological tissue and improve blood circulation.
Our observations show that effective long-term treatment of almost all diseases is impossible without normalizing gut function.
Functions of Gut Microflora
Normal gut microflora produces essential vitamins and enzymes, determines the barrier function of the intestines, and regulates antibody production in the intestinal lymph nodes. Disruptions in its composition or localization can lead to diseases and weakened immunity.
The Challenge of Eliminating Pathogenic Infections
A key goal of medical treatment is to eliminate harmful infections while preserving beneficial microflora. Antibiotics destroy both pathogenic and beneficial bacteria, while the GI emitter emits infrared (IR) energy in a specific spectrum that selectively eliminates only pathogenic bacteria while preserving beneficial microflora. This is because beneficial bacteria have an absorption spectrum close to that of human cells, and the IR radiation exerts a bacteriostatic rather than a bactericidal effect on them.
The Balance of Microorganisms in the Human Body
The human body coexists with many microorganisms, which often provide benefits rather than harm. For example, lactic acid bacteria aid in digesting food residues. Different bacterial species compete with each other, helping to control pathogenic bacteria and prevent overgrowth.
Pathogenic bacteria are the exception, not the rule, and even within a single bacterial species, some strains can be harmful while others are harmless. The concept of pathogenicity is relative, yet certain pathogenic microorganisms exist and can damage tissues, leading to disease. Most microorganisms are conditionally pathogenic, such as tuberculosis bacteria, which do not always cause illness.
The Role of Candida and Other Opportunistic Microorganisms
Under certain conditions, harmless microorganisms can become pathogenic. For example, the Candida fungus, a normal part of oral flora, can overgrow when antibiotics eliminate competing bacteria, leading to severe fungal infections in the digestive tract.
The Role of Microcirculation in Disease DevelopmentAnother factor increasing the risk of toxicity and disease is impaired microcirculation, which leads to blood and lymph stagnation. This disrupts the nutritional supply of organs and tissues and impairs waste elimination, causing intoxication. Since all nutrients and energy sources come from the blood and metabolic waste is removed via blood and lymph, normalizing circulation is critical for recovery.
Restoring Gut Microflora with the GI Emitter
The GI emitter is used on the abdominal area to restore gut microflora. The procedure involves:
One of the essential conditions for maintaining health is a balanced gut microflora. In all patients, regardless of the disease—whether oncological, cardiovascular, allergic, endocrine, dermatological, wound infections, mastitis, peritonitis, gangrene, and others—there is a disruption of the microflora in the small and large intestines. Often, it is impossible to restore the balance of the autonomic nervous system without normalizing intestinal function.
After chemotherapy or radiation therapy, gut microflora is completely disrupted, leading to dysfunction of the intestines. The use of the GI emitter in such cases helps restore intestinal function within 5–15 days after chemotherapy (with three daily 10-minute sessions) and 30–90 days after radiation therapy under the same regimen. Patients who have not undergone other treatments can fully restore normal intestinal function within 1–2 days.
Among patients with long-term allergic conditions, 95% recovered completely after restoring gut function. Over 50% of patients with psoriasis and other skin conditions also experienced recovery. In cases of hypertension, hypotension, and cholesterol metabolism disorders, 90% of patients recovered after intestinal function normalization and using Z-series emitters to remove pathological tissue and improve blood circulation.
Our observations show that effective long-term treatment of almost all diseases is impossible without normalizing gut function.
Functions of Gut Microflora
Normal gut microflora produces essential vitamins and enzymes, determines the barrier function of the intestines, and regulates antibody production in the intestinal lymph nodes. Disruptions in its composition or localization can lead to diseases and weakened immunity.
The Challenge of Eliminating Pathogenic Infections
A key goal of medical treatment is to eliminate harmful infections while preserving beneficial microflora. Antibiotics destroy both pathogenic and beneficial bacteria, while the GI emitter emits infrared (IR) energy in a specific spectrum that selectively eliminates only pathogenic bacteria while preserving beneficial microflora. This is because beneficial bacteria have an absorption spectrum close to that of human cells, and the IR radiation exerts a bacteriostatic rather than a bactericidal effect on them.
The Balance of Microorganisms in the Human Body
The human body coexists with many microorganisms, which often provide benefits rather than harm. For example, lactic acid bacteria aid in digesting food residues. Different bacterial species compete with each other, helping to control pathogenic bacteria and prevent overgrowth.
Pathogenic bacteria are the exception, not the rule, and even within a single bacterial species, some strains can be harmful while others are harmless. The concept of pathogenicity is relative, yet certain pathogenic microorganisms exist and can damage tissues, leading to disease. Most microorganisms are conditionally pathogenic, such as tuberculosis bacteria, which do not always cause illness.
The Role of Candida and Other Opportunistic Microorganisms
Under certain conditions, harmless microorganisms can become pathogenic. For example, the Candida fungus, a normal part of oral flora, can overgrow when antibiotics eliminate competing bacteria, leading to severe fungal infections in the digestive tract.
The Role of Microcirculation in Disease DevelopmentAnother factor increasing the risk of toxicity and disease is impaired microcirculation, which leads to blood and lymph stagnation. This disrupts the nutritional supply of organs and tissues and impairs waste elimination, causing intoxication. Since all nutrients and energy sources come from the blood and metabolic waste is removed via blood and lymph, normalizing circulation is critical for recovery.
Restoring Gut Microflora with the GI Emitter
The GI emitter is used on the abdominal area to restore gut microflora. The procedure involves:
- Rotating the emitter around the navel while applying gentle abdominal massage.
- The massage is performed in circular motions with light pressure along the intestinal path.
- It is essential to ensure the emitter’s beam is directed at the patient’s abdomen, not the doctor’s hand.
Procedure Protocol for Treating Intestinal Disorders:
1. Procedure Protocol for Restoring Gut MicrofloraGI(s) Emitter Application- Duration: 10–20 minutes per session.
- Frequency: 3 times daily, after meals.
- Purpose: Targets the intestines to restore normal microflora and intestinal function.
2. ZB(s) Emitter Application
- Duration: 10–15 minutes per session.
- Purpose:
- Improves microcirculation in intestinal blood vessels.
- Softens intestinal contents to aid digestion and bowel movements.
- Procedure: Applied immediately after the GI(s) emitter.
- For constipation:
- The procedure may be extended up to 30 minutes.
- Maximum exposure should be directed at the sigmoid colon.
During the process of restoring gut microflora, the use of antibiotics is strictly prohibited, as they destroy beneficial bacteria essential for gut health.
If the patient has pathogenic mycotic flora, treatment is carried out using the AF(s) emitter first, followed by the GI(s) emitter according to point 1.
WOUNDS
Wound Healing and Infection Control
A wound is a mechanical injury to the skin or mucous membrane. Although wounds have been treated since ancient times, there is still no universally accepted theory of wound healing. A century ago, microorganisms were identified in wounds, and since then, wound suppuration has been considered an infectious complication. Therefore, the same measures used to prevent and treat infectious diseases should be applied—protecting the body from contact with pathogens and eliminating microbes.
Antisepsis played a crucial role in the development of modern surgery. Sterile instruments, surgical linens, hand disinfection, and preoperative skin preparation significantly reduced the number of postoperative infections. Many bactericidal agents were introduced, but they failed to meet expectations when treating contaminated, already-infected accidental wounds.
The discovery of sulfa drugs and antibiotics renewed hope for overcoming infections. Initially, their use led to a significant reduction in postoperative complications, especially fatal ones. However, over time, as these drugs were increasingly used, infection rates returned to their original levels, and the mortality rate from sepsis has now reached 80%.
It is important to recognize that infectious agents continuously mutate and adapt to external influences. In the past, boiling was sufficient for sterilization, but today even exposure to steam at 132°C or dry heat at 160–180°C does not guarantee complete sterilization. Additionally, such treatments cause instrument corrosion, leading to undesirable consequences. Other factors, such as environmental degradation, reduced physical activity leading to poor circulation and cholesterol metabolism disorders, unbalanced diets, and disruptions in gut microbiota, further contribute to a sharp decline in immunity.
In recent years, reports have increasingly highlighted the insufficient effectiveness of antibiotics in preventing wound infections. These drugs are toxic, can cause severe allergic reactions, and suppress the immune system, increasing the risk of severe sepsis.
The human body maintains a balance between bacterial and fungal microflora. Bacteria control fungal growth, while fungi produce antibiotics that inhibit bacteria. The widespread use of antibiotics has disrupted this balance, favoring fungal overgrowth, which has led to the rise of hard-to-treat fungal infections. Initially, new antibiotics show strong effects, as they target pathogenic bacteria. However, with repeated use, the microbial balance shifts toward fungi, making infections more severe and resistant to treatment. The immune system must then fight not bacteria but fungi, compensating for fungal toxins. The body's method of detoxification depends on individual drainage pathways—skin conditions like psoriasis and eczema occur when toxins are expelled through the skin, while gastrointestinal issues arise when toxins are processed through digestion. Periodontal disease is also often linked to this drainage process.
Example: Psoriasis as a Skin Drainage Mechanism
Initially, in psoriasis, bacterial byproducts are expelled, and antibiotic treatment provides a noticeable improvement by eliminating pathogenic bacteria. However, when psoriasis reoccurs, the same antibiotic worsens the condition instead of improving it. This is because fungal overgrowth has replaced bacterial infections, and there are no bacteria left to counteract fungal proliferation.
Treating Severe Psoriasis
Given the declining effectiveness of antibiotics, surgeons have returned to using antiseptics such as organic and inorganic acids, potassium permanganate, and silver nitrate solutions.
Suppuration and Infectious Diseases
Suppuration resembles infectious diseases like dysentery in that it involves microbes and can spread in hospital settings. However, there are key differences:
Microbial Ecology in Wounds
When a wound occurs, various microbes enter, but over time, the wound's microbial flora shifts to resemble that of the skin, mucous membranes, and intestines—Staphylococcus, Pseudomonas aeruginosa, and anaerobes. Only microbes that thrive in the wound's specific physical-chemical conditions survive.
A key characteristic of wound-colonizing microbes is their powerful enzymatic system capable of breaking down proteins. In the intestines, these microbes hydrolyze food residues for nutrient absorption. In wounds, they help break down necrotic tissue, which must be removed for healing. While the body produces most of the necessary enzymes, microbes assist in this process.
Thus, microbes in wounds facilitate the breakdown of dead tissue and accelerate healing. Completely eliminating them would prevent necrotic tissue removal, counteracting natural healing mechanisms. Antibiotics and sulfa drugs disrupt the ecological relationship between microbes and wounds, interfering with evolutionary processes.
Understanding Wound Healing
Recent research has shed light on wound healing processes, confirming that all wounds heal through inflammation, and most, if not surgically treated, undergo suppuration.
Immediately after an injury, a neurovascular reaction occurs, leading to swelling. Tissue acidity increases, with pH dropping below 5. Osmotic pressure rises, drawing in plasma and blood cells (erythrocytes, leukocytes, platelets). Neutrophils and eosinophils release proteolytic enzymes that break down proteins, aiding in the removal of dead tissue. Living cells contain anti-enzymes to prevent excessive breakdown.
Traditionally, these changes were considered pathological—acidosis was blamed on metabolic disturbances, and tissue swelling on microcirculation failure. However, these processes occur universally in wounds and serve essential functions:
Inflammation and suppuration in wounds accelerate cleansing and healing. A wound cannot close while necrotic tissue remains. If there is little dead tissue, macrophages remove it and the wound heals. However, when necrotic tissue is abundant, microbes proliferate and form pus, triggering further immune responses.
Surgical debridement is not always possible, and even when performed, it may not remove all necrotic tissue. Therefore, preventing suppuration and developing non-surgical wound treatment methods remain ongoing challenges.
Conclusion
To optimize wound healing and infection control, a more comprehensive approach is needed—one that integrates:
A wound is a mechanical injury to the skin or mucous membrane. Although wounds have been treated since ancient times, there is still no universally accepted theory of wound healing. A century ago, microorganisms were identified in wounds, and since then, wound suppuration has been considered an infectious complication. Therefore, the same measures used to prevent and treat infectious diseases should be applied—protecting the body from contact with pathogens and eliminating microbes.
Antisepsis played a crucial role in the development of modern surgery. Sterile instruments, surgical linens, hand disinfection, and preoperative skin preparation significantly reduced the number of postoperative infections. Many bactericidal agents were introduced, but they failed to meet expectations when treating contaminated, already-infected accidental wounds.
The discovery of sulfa drugs and antibiotics renewed hope for overcoming infections. Initially, their use led to a significant reduction in postoperative complications, especially fatal ones. However, over time, as these drugs were increasingly used, infection rates returned to their original levels, and the mortality rate from sepsis has now reached 80%.
It is important to recognize that infectious agents continuously mutate and adapt to external influences. In the past, boiling was sufficient for sterilization, but today even exposure to steam at 132°C or dry heat at 160–180°C does not guarantee complete sterilization. Additionally, such treatments cause instrument corrosion, leading to undesirable consequences. Other factors, such as environmental degradation, reduced physical activity leading to poor circulation and cholesterol metabolism disorders, unbalanced diets, and disruptions in gut microbiota, further contribute to a sharp decline in immunity.
In recent years, reports have increasingly highlighted the insufficient effectiveness of antibiotics in preventing wound infections. These drugs are toxic, can cause severe allergic reactions, and suppress the immune system, increasing the risk of severe sepsis.
The human body maintains a balance between bacterial and fungal microflora. Bacteria control fungal growth, while fungi produce antibiotics that inhibit bacteria. The widespread use of antibiotics has disrupted this balance, favoring fungal overgrowth, which has led to the rise of hard-to-treat fungal infections. Initially, new antibiotics show strong effects, as they target pathogenic bacteria. However, with repeated use, the microbial balance shifts toward fungi, making infections more severe and resistant to treatment. The immune system must then fight not bacteria but fungi, compensating for fungal toxins. The body's method of detoxification depends on individual drainage pathways—skin conditions like psoriasis and eczema occur when toxins are expelled through the skin, while gastrointestinal issues arise when toxins are processed through digestion. Periodontal disease is also often linked to this drainage process.
Example: Psoriasis as a Skin Drainage Mechanism
Initially, in psoriasis, bacterial byproducts are expelled, and antibiotic treatment provides a noticeable improvement by eliminating pathogenic bacteria. However, when psoriasis reoccurs, the same antibiotic worsens the condition instead of improving it. This is because fungal overgrowth has replaced bacterial infections, and there are no bacteria left to counteract fungal proliferation.
Treating Severe Psoriasis
Given the declining effectiveness of antibiotics, surgeons have returned to using antiseptics such as organic and inorganic acids, potassium permanganate, and silver nitrate solutions.
Suppuration and Infectious Diseases
Suppuration resembles infectious diseases like dysentery in that it involves microbes and can spread in hospital settings. However, there are key differences:
- Infectious diseases have specific causative agents, whereas different wounds contain different microbial species.
- The presence of microbes in a wound does not always lead to suppuration. In surgical wound swabs, microbes were found in 80–90% of cases, but actual postoperative infections were much lower.
- During World War II, surgeons successfully sutured infected wounds, and four out of five healed without complications.
Microbial Ecology in Wounds
When a wound occurs, various microbes enter, but over time, the wound's microbial flora shifts to resemble that of the skin, mucous membranes, and intestines—Staphylococcus, Pseudomonas aeruginosa, and anaerobes. Only microbes that thrive in the wound's specific physical-chemical conditions survive.
A key characteristic of wound-colonizing microbes is their powerful enzymatic system capable of breaking down proteins. In the intestines, these microbes hydrolyze food residues for nutrient absorption. In wounds, they help break down necrotic tissue, which must be removed for healing. While the body produces most of the necessary enzymes, microbes assist in this process.
Thus, microbes in wounds facilitate the breakdown of dead tissue and accelerate healing. Completely eliminating them would prevent necrotic tissue removal, counteracting natural healing mechanisms. Antibiotics and sulfa drugs disrupt the ecological relationship between microbes and wounds, interfering with evolutionary processes.
Understanding Wound Healing
Recent research has shed light on wound healing processes, confirming that all wounds heal through inflammation, and most, if not surgically treated, undergo suppuration.
Immediately after an injury, a neurovascular reaction occurs, leading to swelling. Tissue acidity increases, with pH dropping below 5. Osmotic pressure rises, drawing in plasma and blood cells (erythrocytes, leukocytes, platelets). Neutrophils and eosinophils release proteolytic enzymes that break down proteins, aiding in the removal of dead tissue. Living cells contain anti-enzymes to prevent excessive breakdown.
Traditionally, these changes were considered pathological—acidosis was blamed on metabolic disturbances, and tissue swelling on microcirculation failure. However, these processes occur universally in wounds and serve essential functions:
- Acidification inhibits pathogenic bacteria and fungal growth.
- Increased osmotic pressure helps remove dead cells from deep tissues.
- Enhanced enzymatic activity accelerates tissue breakdown and clearance (e.g., pepsin is most active at pH 3).
Inflammation and suppuration in wounds accelerate cleansing and healing. A wound cannot close while necrotic tissue remains. If there is little dead tissue, macrophages remove it and the wound heals. However, when necrotic tissue is abundant, microbes proliferate and form pus, triggering further immune responses.
Surgical debridement is not always possible, and even when performed, it may not remove all necrotic tissue. Therefore, preventing suppuration and developing non-surgical wound treatment methods remain ongoing challenges.
Conclusion
To optimize wound healing and infection control, a more comprehensive approach is needed—one that integrates:
- Reducing antibiotic overuse to prevent microbial imbalance.
- Utilizing antiseptics like organic acids and silver compounds.
- Supporting immune function through proper nutrition and gut microbiome health.
- Recognizing inflammation as a natural healing process and avoiding interventions that disrupt it.
By understanding the body's natural defense mechanisms, medical practices can evolve toward more effective and sustainable wound care strategies.
- Accelerate the breakdown of necrotic tissues,
- Preserve the ability of tissues to regenerate and inhibit microbial growth.
When using resonant IR emitters, treatment is more effective and prevents suppuration. Even if a wound has already become infected, it can still heal quickly and without negative consequences.
The primary emitter for wound treatment is the GI series, as it has anti-inflammatory and anti-infective properties. Additionally, it helps normalize intestinal microflora, including the beneficial bacteria necessary for wound healing without suppuration.
As previously mentioned, proper blood circulation is essential. The GI emitter reduces inflammatory reactions, thereby improving circulation.
A key factor in this process is corticosterone. Its deoxy form triggers inflammation, while its oxy form reduces it. The GI emitter facilitates the conversion of deoxy-corticosterone into its oxy form.
Furthermore, the GI emitter promotes the dissociation of transcortin into cortisol, which, under these conditions, supports catabolic reactions—specifically, the breakdown of necrotic tissue into smaller protein fragments and even amino acids that the body can reuse. Additionally, transcortin releases vital blood proteins needed by the body at that moment.
To further restore blood circulation, Z-series emitters can be used. After wound healing, exposure to this emitter can help remove keloid tissue, making it useful for plastic surgery applications as well.
In any case, K-series emitters should also be used to normalize immune system function by regulating metabolic processes. Additionally, during illness, the body predominantly relies on glycolytic energy metabolism, placing extra strain on the pancreas. K-series emitters help restore endocrine system function.
The primary emitter for wound treatment is the GI series, as it has anti-inflammatory and anti-infective properties. Additionally, it helps normalize intestinal microflora, including the beneficial bacteria necessary for wound healing without suppuration.
As previously mentioned, proper blood circulation is essential. The GI emitter reduces inflammatory reactions, thereby improving circulation.
A key factor in this process is corticosterone. Its deoxy form triggers inflammation, while its oxy form reduces it. The GI emitter facilitates the conversion of deoxy-corticosterone into its oxy form.
Furthermore, the GI emitter promotes the dissociation of transcortin into cortisol, which, under these conditions, supports catabolic reactions—specifically, the breakdown of necrotic tissue into smaller protein fragments and even amino acids that the body can reuse. Additionally, transcortin releases vital blood proteins needed by the body at that moment.
To further restore blood circulation, Z-series emitters can be used. After wound healing, exposure to this emitter can help remove keloid tissue, making it useful for plastic surgery applications as well.
In any case, K-series emitters should also be used to normalize immune system function by regulating metabolic processes. Additionally, during illness, the body predominantly relies on glycolytic energy metabolism, placing extra strain on the pancreas. K-series emitters help restore endocrine system function.
TYPES OF ENERGY METABOLISM AND ITS SIGNIFICANCE IN DISEASE TREATMENT
The human body exhibits four types of energy metabolism:
- Anaerobic creatine phosphate
- Anaerobic glycolytic
- Aerobic using glucose
- Aerobic using fatty acids
The creatine phosphate cycle is very short, lasting about 6-9 seconds. This cycle occurs anaerobically due to the breakdown of creatine phosphate and is accompanied by the release of the maximum amount of energy per unit of time. This energy exchange pathway is utilized during activities of very high intensity (sprinting, high jumping, weightlifting, etc.).
Aerobic glycolytic metabolism occurs constantly in the body with the participation of oxygen.
Anaerobic glycolytic metabolism occupies an intermediate position between the previous two types of metabolism and proceeds through the breakdown of glycogen. This type of energy exchange is predominantly used by the body in the presence of pathology. Given that maintaining the activity of this metabolic pathway requires active functioning of the pancreas, it is essential to conduct therapy aimed at enhancing its functional activity.
Aerobic metabolism through fatty acids also occurs constantly with the participation of oxygen.
It should be noted that in aerobic metabolism, one molecule of glucose produces 38 molecules of ATP, whereas in anaerobic metabolism, only 2 ATP molecules are produced. The breakdown of glucose is carried out by 11 enzymes. As mentioned earlier, enzymes are highly specific. In particular, at a certain pH, they facilitate one set of reactions, while at a different pH, they may activate other reactions, including reverse ones. This principle applies to enzymes involved in energy metabolism as well. As glucose is consumed, the blood becomes increasingly acidic due to metabolic byproducts. During aerobic activity, this occurs mainly due to carbonic acid, while during anaerobic metabolism, it is due to the accumulation of lactic acid. When a certain pH threshold is reached, enzymes can no longer break down glucose, which is perceived as fatigue. Under these conditions, reactions proceed in the reverse direction—glucose is synthesized from lactic acid molecules, ensuring maximum efficiency in the utilization of energy substrates.
It is well known that glucose is an essential dietary component. Its oxidation releases between one-third and one-half of the energy used in the body. Another important energy resource is fat, but the roles of glucose and fats in energy metabolism vary across different organs. The heart, for example, exclusively uses fatty acids—products of fat breakdown—as fuel. Skeletal muscles require glucose only for activation, but they function more efficiently using fatty acids as their energy source. However, nerve cells, including those in the brain, rely exclusively on glucose through aerobic metabolism. Their energy demand constitutes 20-30% of the total energy balance, accounting for more than 50% of total glucose consumption. Nerve cells require a constant supply of glucose every second.
During respiration, humans absorb only 3-7% of inhaled oxygen. As mentioned earlier, the brain and heart can function only in the presence of oxygen. If oxygen is insufficient for any reason, it is stored in the form of superoxide:
Decomposition can also follow another pathway:
It can also attack blood vessels in the brain, potentially leading to a hemorrhagic stroke.
A third possible pathway of decomposition is:
The venom of many snakes, particularly cobras, consists of phospholipases. Their mechanism of action involves breaking down cell membranes, essentially causing an "infarction" of the cells that interact with this enzyme.
Given the above, it is necessary to stimulate the pancreas, as the function of the nervous system plays a crucial role in recovery and preventive protection of the body.
Pancreatic function stimulation is carried out through exposure to radiation generated by KL. The emitter is directed at the pancreas from three angles—at the head, body, and tail.
The exposure duration is 5 minutes from each angle. The procedure can be performed multiple times per day.
When targeting the pancreas, attention should be paid to the condition of its ducts. If any abnormalities are present, an ultrasound examination is recommended, along with selecting the appropriate emitter (RC, RV, GI, ZB) for restoration.
Aerobic glycolytic metabolism occurs constantly in the body with the participation of oxygen.
Anaerobic glycolytic metabolism occupies an intermediate position between the previous two types of metabolism and proceeds through the breakdown of glycogen. This type of energy exchange is predominantly used by the body in the presence of pathology. Given that maintaining the activity of this metabolic pathway requires active functioning of the pancreas, it is essential to conduct therapy aimed at enhancing its functional activity.
Aerobic metabolism through fatty acids also occurs constantly with the participation of oxygen.
It should be noted that in aerobic metabolism, one molecule of glucose produces 38 molecules of ATP, whereas in anaerobic metabolism, only 2 ATP molecules are produced. The breakdown of glucose is carried out by 11 enzymes. As mentioned earlier, enzymes are highly specific. In particular, at a certain pH, they facilitate one set of reactions, while at a different pH, they may activate other reactions, including reverse ones. This principle applies to enzymes involved in energy metabolism as well. As glucose is consumed, the blood becomes increasingly acidic due to metabolic byproducts. During aerobic activity, this occurs mainly due to carbonic acid, while during anaerobic metabolism, it is due to the accumulation of lactic acid. When a certain pH threshold is reached, enzymes can no longer break down glucose, which is perceived as fatigue. Under these conditions, reactions proceed in the reverse direction—glucose is synthesized from lactic acid molecules, ensuring maximum efficiency in the utilization of energy substrates.
It is well known that glucose is an essential dietary component. Its oxidation releases between one-third and one-half of the energy used in the body. Another important energy resource is fat, but the roles of glucose and fats in energy metabolism vary across different organs. The heart, for example, exclusively uses fatty acids—products of fat breakdown—as fuel. Skeletal muscles require glucose only for activation, but they function more efficiently using fatty acids as their energy source. However, nerve cells, including those in the brain, rely exclusively on glucose through aerobic metabolism. Their energy demand constitutes 20-30% of the total energy balance, accounting for more than 50% of total glucose consumption. Nerve cells require a constant supply of glucose every second.
During respiration, humans absorb only 3-7% of inhaled oxygen. As mentioned earlier, the brain and heart can function only in the presence of oxygen. If oxygen is insufficient for any reason, it is stored in the form of superoxide:
R1OOR2, which, upon decomposition:
2R1OOR2 → 2R1OR2 + O2
releases additional oxygen necessary for the normal functioning of the heart and nervous system.Decomposition can also follow another pathway:
R1OOR2 → R1OО• + O•
This process forms a superoxide radical R1OО•. Such a radical can attack the membranes of heart muscle cells (myocardium), ultimately leading to cell fusion. This, in turn, causes a heart attack.It can also attack blood vessels in the brain, potentially leading to a hemorrhagic stroke.
A third possible pathway of decomposition is:
R1OOR2 → 2R1O•
Such radicals often lead to oncological diseases. Thus, hypoxia is at the core of the most severe diseases. Fortunately, in many cases, the nervous system is capable of protecting the body.The venom of many snakes, particularly cobras, consists of phospholipases. Their mechanism of action involves breaking down cell membranes, essentially causing an "infarction" of the cells that interact with this enzyme.
Given the above, it is necessary to stimulate the pancreas, as the function of the nervous system plays a crucial role in recovery and preventive protection of the body.
Pancreatic function stimulation is carried out through exposure to radiation generated by KL. The emitter is directed at the pancreas from three angles—at the head, body, and tail.
The exposure duration is 5 minutes from each angle. The procedure can be performed multiple times per day.
When targeting the pancreas, attention should be paid to the condition of its ducts. If any abnormalities are present, an ultrasound examination is recommended, along with selecting the appropriate emitter (RC, RV, GI, ZB) for restoration.
INTERCONNECTION OF ORGANS AND TISSUES
In some cases, biochemical processes in cells are highly specialized, and their functions are quite limited. A good example of this is erythrocytes, where only anaerobic glucose catabolism takes place. In other cases, cells exhibit multipotency, meaning they have the ability to carry out a wide variety of enzymatic transformations. For instance, hepatocytes participate in anabolic and catabolic processes, interconversions of carbohydrates, fats, and proteins, as well as other metabolic functions.
Metabolic processes are primarily carried out by the liver. Some processes also take place in other organs and tissues. For example, glucose dephosphorylation and gluconeogenesis occur to a lesser extent in the kidneys, lipogenesis takes place in adipose tissue, cholesterol synthesis in the mucous membrane of the gastrointestinal tract, heme synthesis in the reticuloendothelial system, aromatic amino acid metabolism in nervous tissue, as well as purine and pyrimidine metabolism or methyl group transfer in certain other tissues.
All cells of an organism contain the same DNA molecules. At a certain stage of each cell's life, DNA expression is ensured either through replication during cell division or through transcription during protein synthesis in the growth phase. As cells differentiate and acquire their final specialized forms in the organs of our body, they gradually lose (to varying degrees) the ability to express genes, and their DNA gradually becomes "silent." In extreme cases, DNA may disappear entirely—this happens, for example, in erythrocytes. Another example is the loss of replication ability, as observed in mature neurons, which do not divide in adults.
In many cell types, only a portion of the total DNA undergoes transcription, meaning it gives rise to messenger RNA (mRNA) corresponding to enzymes and other proteins specific to that cell. In other cell types, reading DNA to generate certain informational sequences is impossible unless a special substance—the repressor—is removed. The degree and timing of these complex regulatory factors determine the biochemical specialization of tissues and the development of organs. As differentiated tissues partially lose their metabolic capabilities during specialization, they become dependent on other cell types for the supply of essential metabolites and the removal of metabolic waste products.
Metabolism, from the moment nutrients enter the body, includes the stages of digestion and absorption in the gastrointestinal tract. Naturally, these processes are followed by the transport of nutrients through the main "gateway"—the aptly named "portal vein"—to the liver. Emphasizing the liver’s role in metabolism is appropriate given its high metabolic activity as the body's primary "chemical factory." No other organ matches the liver in diversity and adaptability, as it conducts complex, interconnected metabolic processes that influence the entire organism. Let us now summarize these processes in relation to the functions of other organs.
In many cell types, only a portion of the total DNA undergoes transcription, meaning it gives rise to messenger RNA (mRNA) corresponding to enzymes and other proteins specific to that cell. In other cell types, reading DNA to generate certain informational sequences is impossible unless a special substance—the repressor—is removed. The degree and timing of these complex regulatory factors determine the biochemical specialization of tissues and the development of organs. As differentiated tissues partially lose their metabolic capabilities during specialization, they become dependent on other cell types for the supply of essential metabolites and the removal of metabolic waste products.
Metabolism, from the moment nutrients enter the body, includes the stages of digestion and absorption in the gastrointestinal tract. Naturally, these processes are followed by the transport of nutrients through the main "gateway"—the aptly named "portal vein"—to the liver. Emphasizing the liver’s role in metabolism is appropriate given its high metabolic activity as the body's primary "chemical factory." No other organ matches the liver in diversity and adaptability, as it conducts complex, interconnected metabolic processes that influence the entire organism. Let us now summarize these processes in relation to the functions of other organs.
LIVER
The liver functions as a gland with both exocrine and endocrine roles. The product of external secretion (exocrine function) is bile, which is released by the liver into the gastrointestinal tract. Bile is a solution containing metabolic waste products such as bile pigments, as well as essential fat digestion accelerators—bile acid salts. These salts are the primary oxidation products of steroids. The reabsorption of bile acid salts from the gastrointestinal tract is an important self-regulating mechanism operating on a feedback principle since the oxidation of cholesterol in liver cells is inhibited by bile acid salts returning from the intestine.
The level of cholesterol biosynthesis occurring in the gastrointestinal tract is also crucial. This process is regulated by a feedback mechanism, as cholesterol biosynthesis is largely inhibited by bile acid salts, which are absorbed by the mucosal cells.
The level of cholesterol biosynthesis occurring in the gastrointestinal tract is also crucial. This process is regulated by a feedback mechanism, as cholesterol biosynthesis is largely inhibited by bile acid salts, which are absorbed by the mucosal cells.
The products of the liver's internal secretion (endocrine function) are not hormones but metabolites that circulate in the bloodstream and influence the functions of other cells. These metabolites include:
- Glucose, secreted primarily during fasting under the stimulation of glucocorticoids or glucagon, as well as during intense muscle activity. This ensures the needs of glycolysis in brain and muscle tissues.
- Triglycerides, released after carbohydrate intake in the gastrointestinal tract or under insulin stimulation, primarily contributing to lipogenesis processes in adipose tissue.
- Ketone bodies, produced in excess during fasting or when consuming a diet rich in fats and low in carbohydrates. These compounds serve as an energy source for muscle and nervous tissue.
Of course, these are just a few general pathways through which liver-produced metabolites influence biochemical reactions in other tissues. Additionally, the liver is responsible for the synthesis and secretion of albumin, serum lipoproteins, blood clotting factors, and other important nitrogenous metabolic products utilized in various tissues.
INTERCONNECTION BETWEEN METABOLIC PROCESSES IN THE LIVER AND OTHER TISSUES
Extratonic tissues exhibit a certain degree of metabolic autonomy. The main reactions leading to energy production (glycolysis, citric acid cycle reactions, oxidative phosphorylation, and fatty acid oxidation), as well as key biosynthetic processes (protein synthesis, nucleic acid synthesis, lipogenesis, and gluconeogenesis), occur in various cell types, though their activity levels may vary.
Nevertheless, the dependence of these tissues on liver metabolism is often crucial for maintaining normal function and homeostasis. Therefore, it is essential to discuss and summarize some fundamentally important examples of the interrelationships between the liver and other tissues.
ADIPOSE TISSUE AND THE LIVER
The processes of fat metabolism in the liver and adipose tissue are closely interconnected. Triglycerides formed in liver cells serve as an important source of fat stored in adipose depots.
Another source of these fats is newly synthesized triglycerides produced by adipocytes themselves. Since glucose provides carbon atoms for the fatty acids and glycerol residues synthesized in adipose tissue, and the liver plays a key role in regulating blood glucose levels, lipogenesis in fat depots is doubly dependent on liver metabolism.
Another source of these fats is newly synthesized triglycerides produced by adipocytes themselves. Since glucose provides carbon atoms for the fatty acids and glycerol residues synthesized in adipose tissue, and the liver plays a key role in regulating blood glucose levels, lipogenesis in fat depots is doubly dependent on liver metabolism.
During periods of abundant nutrition, the flow of carbon atoms from liver cells to adipose tissue shifts in the opposite direction when energy intake decreases. In such cases, the activation of hormone-sensitive lipase in adipocytes leads to the release of glycerol and fatty acids into the bloodstream. These triglyceride breakdown products help meet the liver’s energy needs during fasting, as fatty acid oxidation produces NADH and ATP required for gluconeogenesis. On the other hand, fatty acids can be directly used as an alternative to glucose in extrahepatic tissues such as muscle.
MUSCLES AND THE LIVER
There is a direct metabolic interconnection between muscle and liver tissues on multiple levels. During periods of abundant nutrition, both tissues extract glucose from the bloodstream, leading to glycogen storage. Intense physical activity, a key stimulus for glycogenolysis in muscles, results in glycogen breakdown into lactate, which diffuses into the blood. Conversely, reduced food intake primarily activates glycogen breakdown in the liver, but only to release glucose into circulation.
This difference is also evident in the balance between glycolysis and gluconeogenesis. In muscle tissue, where phosphofructokinase and pyruvate kinase activities are high but fructose-bisphosphatase, pyruvate carboxylase, and phosphoenolpyruvate carboxykinase activities are low, glucose metabolism leans toward glycolysis, generating ATP and lactate. In contrast, the liver, with an opposite enzyme activity ratio, favors gluconeogenesis, regenerating glucose from incoming lactate using ATP derived from fatty acid oxidation to meet muscle energy demands.
During prolonged fasting, when muscle proteins undergo catabolism, skeletal muscles convert significant amounts of pyruvate into alanine via transamination. Alanine then serves as a carbon source for gluconeogenesis in the liver, acting as an alternative to lactate in the glucose-lactate cycle, which involves both the liver and skeletal muscles. It is important to note that cardiac muscle is an aerobic tissue, rich in mitochondria, and equipped with a lactate dehydrogenase system that prefers to consume lactate rather than produce it.
Both skeletal and cardiac muscles rely on the liver by utilizing ketone bodies—beta-hydroxybutyrate and acetoacetate. Since the liver lacks the enzyme necessary to activate acetoacetate into its CoA derivative, ketone bodies enter the bloodstream in large amounts during low-calorie, carbohydrate-poor, or fat-rich diets, when hepatic fatty acid oxidation exceeds its acetyl-CoA disposal capacity. Muscles, kidneys, and other extrahepatic tissues activate acetoacetate using succinyl-CoA and extract energy by oxidizing the resulting acetyl-CoA.
This difference is also evident in the balance between glycolysis and gluconeogenesis. In muscle tissue, where phosphofructokinase and pyruvate kinase activities are high but fructose-bisphosphatase, pyruvate carboxylase, and phosphoenolpyruvate carboxykinase activities are low, glucose metabolism leans toward glycolysis, generating ATP and lactate. In contrast, the liver, with an opposite enzyme activity ratio, favors gluconeogenesis, regenerating glucose from incoming lactate using ATP derived from fatty acid oxidation to meet muscle energy demands.
During prolonged fasting, when muscle proteins undergo catabolism, skeletal muscles convert significant amounts of pyruvate into alanine via transamination. Alanine then serves as a carbon source for gluconeogenesis in the liver, acting as an alternative to lactate in the glucose-lactate cycle, which involves both the liver and skeletal muscles. It is important to note that cardiac muscle is an aerobic tissue, rich in mitochondria, and equipped with a lactate dehydrogenase system that prefers to consume lactate rather than produce it.
Both skeletal and cardiac muscles rely on the liver by utilizing ketone bodies—beta-hydroxybutyrate and acetoacetate. Since the liver lacks the enzyme necessary to activate acetoacetate into its CoA derivative, ketone bodies enter the bloodstream in large amounts during low-calorie, carbohydrate-poor, or fat-rich diets, when hepatic fatty acid oxidation exceeds its acetyl-CoA disposal capacity. Muscles, kidneys, and other extrahepatic tissues activate acetoacetate using succinyl-CoA and extract energy by oxidizing the resulting acetyl-CoA.
Even at rest, there is a continuous flow of acetoacetate and other ketone bodies from the liver to peripheral tissues, where they contribute significantly to energy production, particularly heat generation. However, while cardiac and skeletal muscles depend on liver-derived metabolites, they primarily satisfy their ATP needs through the oxidation of fatty acids released from adipose tissue stores.
KIDNEYS AND THE LIVER
The process of gluconeogenesis occurs in both the kidneys and the liver, but the kidneys generate only a small fraction of the total glucose (approximately one-tenth). However, in cases of liver dysfunction or under conditions of acidosis, where there is an increased breakdown of amino acids into alpha-keto acids—precursors of glucose in the kidneys—their contribution to gluconeogenesis significantly increases.
The kidneys rely on the liver for the supply of glutamine, which serves as a source of ammonia necessary for neutralizing excreted hydrogen ions. On the other hand, liver function depends on the excretory role of the kidneys, which remove urea and other metabolic waste products from the bloodstream while preserving essential liver-produced substances such as glucose, amino acids, and proteins.
BRAIN AND LIVER
Finally, the metabolic processes in brain tissues and the liver are closely interconnected. This is primarily because nervous tissue is entirely dependent on the continuous supply of glucose, which is ensured by the liver. During the catabolism of glucose, the following are produced:
- Energy required by brain cells for the active transport of ions involved in excitation processes;
- Acetyl-CoA for the synthesis of lipids, myelin, and acetylcholine;
- Material for the formation of carbon chains of glutamate, gamma-aminobutyric acid (GABA), and other amino acids.
Since active amino acid metabolism occurs in brain tissues, and the brain is particularly sensitive to the toxic effects of ammonia, effective protective mechanisms have developed to bind NH₃. This primarily occurs through the formation of glutamate and glutamine. Ultimately, the liver removes NH₃ from brain tissues and other peripheral tissues by converting it into urea.
Liver dysfunction or metabolic shifts that disrupt the urea cycle reactions significantly impact brain development and, consequently, higher nervous activity. This is due to the brain's dependence on the liver for the removal of ammonia and other metabolic end products that are toxic to neural tissue.
Liver dysfunction or metabolic shifts that disrupt the urea cycle reactions significantly impact brain development and, consequently, higher nervous activity. This is due to the brain's dependence on the liver for the removal of ammonia and other metabolic end products that are toxic to neural tissue.
SPINE
SpineAny intoxication—whether caused by infection, toxins, metal ions, gases, stress, etc.—disrupts the circulatory system by causing vascular blockages and hormonal imbalances. This process primarily affects the spine. The inability to transport plastic and energy materials, as well as oxygen, to cells, organs, and tissues leads to brain hypoxia, particularly in the cortex. This, in turn, disrupts the system responsible for regulation, nourishment, and the removal of metabolic waste from organs.
The spine plays a crucial role in human life—it is the foundation of the skeleton, giving the body its shape, keeping it upright, and holding vital organs in place. The spine consists of 24 small vertebrae. Each vertebra has two main parts: the body and the arch. The top and bottom of each vertebra are covered with cartilage. Between the bodies of two vertebrae lies an elastic intervertebral disc, which consists of two hyaline plates, a nucleus pulposus, and a surrounding fibrous ring. The nucleus pulposus contains chondrin, a small number of cartilage cells, and collagen fibers, which give it elasticity. The fibrous ring consists of dense bundles of connective tissue interwoven in different directions.
Intervertebral discs allow the spine to move in various directions and absorb shocks, while the vertebral arches form a canal that houses the spinal cord. The spinal cord serves as the control center for an extensive and complex network of nerves spread throughout the body. This neural network originates from 31 pairs of nerve fibers, each controlling a specific body part and organ. Reflex and autonomic functions of organs are controlled by the spinal cord (except for those regulated by the brain). Pathological processes in the spine lead to compression of nerve fibers and dysfunction of the innervated organs.
Only 1 in 150 middle-aged people has a flexible and, therefore, healthy spine. Spinal disorders, primarily affecting individuals aged 30–50, often result in prolonged disability and, in some cases, permanent impairment.
The most common spinal disease is osteochondrosis, which primarily affects the lumbar-sacral region. This selectivity is likely due to the anatomical and functional peculiarities of the lumbar-sacral area, which evolved as humans adopted an upright posture, significantly increasing the load on the spine—especially the lower back. Additional stress factors include heavy lifting, physical inactivity, and poor nutrition.
Degenerative processes in the intervertebral disc begin with damage to the nucleus pulposus, which dries out, loses its turgor, and undergoes necrosis. This is followed by the breakdown of the fibrous ring’s fibers, progressing from the center outward and fragmenting primarily in its inner layers. The degeneration of the nucleus pulposus leads to a sharp increase in the load on the fibrous ring, causing uneven stretching of its outer sections in areas where resistance is weakest. The resulting bulging narrows the intervertebral foramen. The disc height decreases, causing the upper vertebra to shift slightly backward relative to the lower one, leading to spinal segment instability. The vertical dimensions of the intervertebral foramina shrink. In osteochondrosis, this narrowing can be significantly worsened if a herniated disc develops due to cracks in the fibrous ring.
The narrowing of intervertebral foramina, combined with localized swelling and aseptic inflammation, leads to irritation and compression of spinal roots and nerve bundles, resulting in localized radicular symptoms.
Patients with osteochondrosis typically seek medical attention due to pain syndromes. Pain is usually localized in the lumbar-sacral region (lumbago, lumbalgia) and along the peripheral nerves emerging from the lumbar-sacral plexus, most commonly the sciatic nerve (lumbosacral sciatica). The pain may be unilateral or bilateral, arise suddenly—often due to heavy lifting or abrupt movement—or develop gradually. It can also be triggered by cold exposure. The disease often begins with lumbago syndrome. Chronic cases are characterized by distinct radicular symptoms.
Pain can be sharp, dull, aching, shooting, or burning, intensifying with movement. It triggers reflex muscle tension, causing stiffness and protective postures.
When spinal nerve roots and bundles are affected, the disease leads to muscle hypotrophy, reduced strength, and sensory disturbances. Involvement of the sympathetic nervous system results in decreased skin temperature, trophic disorders, and the formation of trophic ulcers.
The spine plays a crucial role in human life—it is the foundation of the skeleton, giving the body its shape, keeping it upright, and holding vital organs in place. The spine consists of 24 small vertebrae. Each vertebra has two main parts: the body and the arch. The top and bottom of each vertebra are covered with cartilage. Between the bodies of two vertebrae lies an elastic intervertebral disc, which consists of two hyaline plates, a nucleus pulposus, and a surrounding fibrous ring. The nucleus pulposus contains chondrin, a small number of cartilage cells, and collagen fibers, which give it elasticity. The fibrous ring consists of dense bundles of connective tissue interwoven in different directions.
Intervertebral discs allow the spine to move in various directions and absorb shocks, while the vertebral arches form a canal that houses the spinal cord. The spinal cord serves as the control center for an extensive and complex network of nerves spread throughout the body. This neural network originates from 31 pairs of nerve fibers, each controlling a specific body part and organ. Reflex and autonomic functions of organs are controlled by the spinal cord (except for those regulated by the brain). Pathological processes in the spine lead to compression of nerve fibers and dysfunction of the innervated organs.
Only 1 in 150 middle-aged people has a flexible and, therefore, healthy spine. Spinal disorders, primarily affecting individuals aged 30–50, often result in prolonged disability and, in some cases, permanent impairment.
The most common spinal disease is osteochondrosis, which primarily affects the lumbar-sacral region. This selectivity is likely due to the anatomical and functional peculiarities of the lumbar-sacral area, which evolved as humans adopted an upright posture, significantly increasing the load on the spine—especially the lower back. Additional stress factors include heavy lifting, physical inactivity, and poor nutrition.
Degenerative processes in the intervertebral disc begin with damage to the nucleus pulposus, which dries out, loses its turgor, and undergoes necrosis. This is followed by the breakdown of the fibrous ring’s fibers, progressing from the center outward and fragmenting primarily in its inner layers. The degeneration of the nucleus pulposus leads to a sharp increase in the load on the fibrous ring, causing uneven stretching of its outer sections in areas where resistance is weakest. The resulting bulging narrows the intervertebral foramen. The disc height decreases, causing the upper vertebra to shift slightly backward relative to the lower one, leading to spinal segment instability. The vertical dimensions of the intervertebral foramina shrink. In osteochondrosis, this narrowing can be significantly worsened if a herniated disc develops due to cracks in the fibrous ring.
The narrowing of intervertebral foramina, combined with localized swelling and aseptic inflammation, leads to irritation and compression of spinal roots and nerve bundles, resulting in localized radicular symptoms.
Patients with osteochondrosis typically seek medical attention due to pain syndromes. Pain is usually localized in the lumbar-sacral region (lumbago, lumbalgia) and along the peripheral nerves emerging from the lumbar-sacral plexus, most commonly the sciatic nerve (lumbosacral sciatica). The pain may be unilateral or bilateral, arise suddenly—often due to heavy lifting or abrupt movement—or develop gradually. It can also be triggered by cold exposure. The disease often begins with lumbago syndrome. Chronic cases are characterized by distinct radicular symptoms.
Pain can be sharp, dull, aching, shooting, or burning, intensifying with movement. It triggers reflex muscle tension, causing stiffness and protective postures.
When spinal nerve roots and bundles are affected, the disease leads to muscle hypotrophy, reduced strength, and sensory disturbances. Involvement of the sympathetic nervous system results in decreased skin temperature, trophic disorders, and the formation of trophic ulcers.
In severe osteochondrosis with a herniated disc protruding toward the spinal column, the pain syndrome can become persistent.
TREATMENT OF SPINAL DISEASES
For the relief of inflammation and normalization of metabolic processes in the affected area, a "spark" (GI + KL) is prescribed for 20 minutes daily.
After this, a massage is recommended using ZB or ZK lamps "with a tube" along the spine along the paravertebral lines and below the scapular angle along the scapular line.
Normalizing blood circulation along the spine relieves stress on the nervous system, allowing acute conditions to subside in minutes and restoring blood flow to various organs. A few sessions can eliminate chronic conditions.
After this, a massage is recommended using ZB or ZK lamps "with a tube" along the spine along the paravertebral lines and below the scapular angle along the scapular line.
- Massage Techniques for Different ConditionsScoliosis:
- Massage is performed on both sides of the spine using the ZB emitter with light circular motions—10 rotations per point. Finger pressure should not be excessive, as strong pressure can cause inflammation and block microcirculation in the blood vessels supplying the spine. Spinal alignment during this massage occurs due to blood flow normalization, intervertebral disc softening, and the evacuation of metabolic byproducts. The patient's own muscles are used for symmetrical realignment.
- Lordosis or Kyphosis:
- The massage is performed separately—first on one side, then on the other. The patient’s muscles naturally stretch the spine from one side, then the other, leading to alignment. No other treatment method can restore the spine in this way, as traditional traction methods fail to normalize blood flow or restore disc elasticity.
Normalizing blood circulation along the spine relieves stress on the nervous system, allowing acute conditions to subside in minutes and restoring blood flow to various organs. A few sessions can eliminate chronic conditions.
It is recommended to perform the massage 2-3 times per treatment session.
For spinal pathology, treatment should be combined with intestinal function correction using GI + ZB lamps for 20 minutes daily to normalize microflora.
Key Treatment Recommendations:
- Metabolic Process Normalization: Apply KL and ZB lamps to the pancreas for 5 minutes in 3 projections over 5-7 days.
- Detoxification and Vascular Cleansing: Use the Z-series lamp for 20 minutes every other day, combined with the KL(b) lamp to support the body's natural toxin elimination processes.
- Activation of Spinal "Extraordinary Meridians": In the final week of treatment, a "spark" (GI + KL) should be applied using the "CT" setup, directing radiation from the coccyx along the spine.
- Balancing the Autonomic Nervous System: An essential condition is harmonizing the sympathetic and parasympathetic divisions of the autonomic nervous system.
MAINTENANCE OF A STATIONARY STATE
A healthy adult organism is in equilibrium with its surrounding environment. Such equilibrium requires the timely suppression of growth processes, which is just as essential as the ability of cells to grow and divide. Disruptions in normal inhibitory mechanisms can lead to gigantism, obesity, or uncontrolled malignant growth. It is entirely possible that our understanding of malignancy mechanisms in the future will not come from studying the specific causes of rapid growth and aggressiveness in malignant cells, but rather from exploring why normal cells lack these properties.
In an adult organism, the primary factor determining the normal balance of metabolic processes is the relationship between food intake and energy expenditure. Insufficient nutrition quickly triggers a reversible mobilization of the body's energy reserves; however, prolonged malnutrition or starvation leads to irreversible tissue breakdown. Systematic overeating, on the other hand, can result in a pathological condition (obesity) due to the overfilling of tissue depots. Before discussing these two extreme manifestations of metabolic imbalance, let us examine the mechanisms that maintain the consistency of organ and tissue composition, as well as the properties of regulators that influence anabolic and catabolic enzyme activity.
ANABOLIC HORMONES
These agents enhance the growth-promoting effects of excess nutrition.
Growth hormone, a polypeptide secreted by the anterior pituitary gland, stimulates RNA and protein biosynthesis in almost all cells. This overall increase in nitrogenous compound accumulation is accompanied by enhanced uptake of amino acids from circulating blood. One of the factors promoting growth hormone release is an increased level of amino acids in the blood, ensuring maximum enhancement of tissue protein anabolism in the presence of abundant precursors.
Insulin, secreted by the pancreas in response to elevated glucose or amino acid levels in the blood, stimulates the uptake of glucose and amino acids by tissues. Additionally, insulin promotes increased glucose utilization for glycogen synthesis, lipogenesis, and glycolysis while simultaneously suppressing gluconeogenesis in the liver and inhibiting lipolysis in fat depots. This hormone enhances tissue protein anabolism while simultaneously suppressing amino acid catabolism.
Thyroxine, secreted by the thyroid gland in response to thyroid-stimulating hormone (TSH) from the anterior pituitary, stimulates tissue growth and differentiation. This effect primarily manifests in increased protein synthesis, particularly in the formation of mitochondrial oxidative enzymes. Thus, thyroid hormones exert regulatory influence over oxygen consumption and the overall metabolic rate in tissues.
Growth hormone, a polypeptide secreted by the anterior pituitary gland, stimulates RNA and protein biosynthesis in almost all cells. This overall increase in nitrogenous compound accumulation is accompanied by enhanced uptake of amino acids from circulating blood. One of the factors promoting growth hormone release is an increased level of amino acids in the blood, ensuring maximum enhancement of tissue protein anabolism in the presence of abundant precursors.
Insulin, secreted by the pancreas in response to elevated glucose or amino acid levels in the blood, stimulates the uptake of glucose and amino acids by tissues. Additionally, insulin promotes increased glucose utilization for glycogen synthesis, lipogenesis, and glycolysis while simultaneously suppressing gluconeogenesis in the liver and inhibiting lipolysis in fat depots. This hormone enhances tissue protein anabolism while simultaneously suppressing amino acid catabolism.
Thyroxine, secreted by the thyroid gland in response to thyroid-stimulating hormone (TSH) from the anterior pituitary, stimulates tissue growth and differentiation. This effect primarily manifests in increased protein synthesis, particularly in the formation of mitochondrial oxidative enzymes. Thus, thyroid hormones exert regulatory influence over oxygen consumption and the overall metabolic rate in tissues.
Specific anabolic functions are more pronounced in steroid hormones, particularly estrogens, which are produced in the female body (in the ovaries) and stimulate RNA and protein synthesis in target cells (such as the uterus). Male steroid hormones, androgens, synthesized in the testes, induce a similar acceleration of RNA and protein anabolism in various tissues, including skeletal muscles.
CATABOLIC HORMONES
Many hormones participate in regulating processes that compensate for increased energy expenditures during stress or nutritional deficiency.
Glucagon is a polypeptide secreted by the pancreas in response to a sharp decrease in blood glucose levels, counteracting the hypoglycemic effect of insulin. Glucagon specifically enhances glycogenolysis in the liver, leading to an increase in circulating blood glucose. Additionally, glucagon stimulates gluconeogenesis and lipolysis in the liver.
Fatty acids released under the influence of lipase serve as another energy source and contribute to the process of gluconeogenesis.
Catecholamine hormones—norepinephrine, released upon stimulation of sympathetic nerve endings, and adrenaline, secreted by the adrenal medulla—function similarly to glucagon but also affect tissues beyond the liver. They activate glycogenolysis in muscles and the liver, as well as stimulate lipolysis in adipose tissue, leading to increased levels of glucose and fatty acids in plasma.
Glucagon is a polypeptide secreted by the pancreas in response to a sharp decrease in blood glucose levels, counteracting the hypoglycemic effect of insulin. Glucagon specifically enhances glycogenolysis in the liver, leading to an increase in circulating blood glucose. Additionally, glucagon stimulates gluconeogenesis and lipolysis in the liver.
Fatty acids released under the influence of lipase serve as another energy source and contribute to the process of gluconeogenesis.
Catecholamine hormones—norepinephrine, released upon stimulation of sympathetic nerve endings, and adrenaline, secreted by the adrenal medulla—function similarly to glucagon but also affect tissues beyond the liver. They activate glycogenolysis in muscles and the liver, as well as stimulate lipolysis in adipose tissue, leading to increased levels of glucose and fatty acids in plasma.
Glucocorticoid steroid hormones, such as cortisol, are secreted by the adrenal cortex in response to the action of ACTH (adrenocorticotropic hormone) from the anterior pituitary gland. Cortisol counteracts many effects of insulin by blocking glucose uptake by cells and its conversion into fats while also inhibiting protein synthesis in peripheral tissues. At the same time, the liver's production of enzymes involved in amino acid catabolism and gluconeogenesis is stimulated. As a result, protein stores in extrahepatic tissues become depleted due to ongoing protein hydrolysis and suppressed synthesis. Naturally, amino acids flow from peripheral tissues to the liver, increasing both the amount of carbon directed toward glucose production and the amount of nitrogen used for urea formation.
EXHAUSTION OF THE BODY
Starvation refers to the disruption of the intake of essential substances required for the body's metabolic processes (for example, a deficiency of proteins despite an overall excess of food due to carbohydrates or fats, as well as a lack of trace elements, vitamins, and other active substances).
Very often, protein depletion can result from poor nutrition, where essential proteins and fatty acids do not enter the body, even though the food intake is sufficient in caloric value, primarily from carbohydrates. In such cases, we can speak of protein starvation, which may be accompanied by obesity. One can consume all the necessary components in the required amounts and still suffer from protein depletion. This is usually associated with gut microbiota imbalance, which supplies us with structural and energy materials, vitamins, nutrients, antibiotics, etc. If the microbiota fails to ensure the absorption of these substances, even with an excess of essential amino acids in consumed foods, the body will experience a pronounced deficiency of them.
When classifying manifestations of nutritional deficiency, two main conditions are distinguished: kwashiorkor, primarily caused by a protein-deficient diet, and marasmus, a state associated with general protein-caloric malnutrition. Kwashiorkor typically occurs in children aged 1 to 3 years and is characterized by edema, fat retention (especially in the liver, which becomes significantly enlarged), loose skin, sparse hair with no pigment, apathy, and irritability. Marasmus, on the other hand, results in severe growth retardation as early as 6 to 12 months of age. Unlike kwashiorkor-affected children, who often appear bloated and round-faced due to edema, children with marasmus appear withered, with muscle atrophy and no body fat. In classic cases of marasmus, liver, skin, and hair damage are absent: such a child is restless and reactive but not irritable. Between these two extreme conditions, there are many intermediate forms. Practically, it is difficult to distinguish between them, but from a treatment perspective, it is important to determine whether the child’s diet lacked protein, calories, or both.
The underlying principle connecting all these phenomena is based on the idea of energy equivalence between different classes of nutrients and tissue components. When the total calorie intake from primary energy sources (carbohydrates and fats) becomes less than the body's energy expenditure, protein breakdown begins—both dietary and endogenous. This leads to general wasting, affecting muscles and other organs, as observed in marasmus. Thus, starvation can be viewed as a state in which the body "consumes itself" to meet its energy needs. Before this severe and often fatal state occurs, a series of metabolic changes take place, depending on prior nutritional conditions.
Let’s examine the processes occurring in a healthy, well-nourished adult when suddenly deprived of food. Blood glucose levels immediately decrease, leading to reduced insulin secretion and increased glucagon secretion by the pancreas, stimulating hepatic glycogenolysis. If food is absent for 24 hours, liver glycogen stores are largely depleted; by the second day, other mechanisms must be activated. Prolonged glucagon secretion stimulates hormone-sensitive lipase, increasing the release of fatty acids, which are oxidized in the liver. Similarly, ACTH, catecholamines, and other hormones, released in response to stress stimuli affecting regulatory centers, activate adipose tissue lipase. The mobilization of fatty acids from fat stores provides energy to peripheral tissues, the liver, and other internal organs. Unlike short-term energy production from glycogen, triglycerides from fat stores can supply adequate ATP for several weeks.
As the body adapts to prolonged starvation (during the first few weeks), the production of ketone bodies by the liver increases sharply, as fatty acid oxidation predominates, while pyruvate and oxaloacetate levels decrease, limiting the flow of metabolites through the citric acid cycle. After a few weeks, organs such as the heart and, eventually, the brain adjust to meeting a significant portion of their energy needs via ketone bodies. However, if ketone body production exceeds their utilization, metabolic acidosis occurs, which the body attempts to compensate through respiratory CO₂ elimination and renal NH₄⁺ excretion. At the same time, as the body transitions to fatty acid mobilization and oxidation, adipose tissue and the liver progressively deplete key fatty acid biosynthesis enzymes, such as acetyl-CoA carboxylase, fatty acid synthase, and citrate cleavage enzyme.
If starvation persists, tissue proteins are mobilized and used as an energy source. Paradoxically, the first proteins to be broken down are labile proteins of the gastrointestinal tract and digestive enzymes of the pancreas, impairing the utilization of even the small amounts of food that reach the digestive system. Subsequently, proteins from internal organs (liver, spleen) break down, followed by functional muscle proteins, and finally nervous system proteins. This sequence corresponds to normal protein turnover rates in the described metabolic state. Amino acid catabolism, stimulated by elevated glucocorticoid secretion and decreased insulin levels, provides ATP for peripheral tissues and supplies carbon for glucose synthesis in the liver and kidneys.
During the first weeks of starvation, despite an overall increase in protein catabolism, liver enzymes involved in gluconeogenesis, such as aminotransferases, phosphatases, and enzymes catalyzing pyruvate-to-phosphoenolpyruvate conversion, increase significantly. In prolonged starvation, when the brain primarily relies on beta-hydroxybutyrate rather than glucose for energy, gluconeogenesis intensity gradually decreases. Concurrently, muscle mass, overall activity, and energy expenditure decline.
Very often, protein depletion can result from poor nutrition, where essential proteins and fatty acids do not enter the body, even though the food intake is sufficient in caloric value, primarily from carbohydrates. In such cases, we can speak of protein starvation, which may be accompanied by obesity. One can consume all the necessary components in the required amounts and still suffer from protein depletion. This is usually associated with gut microbiota imbalance, which supplies us with structural and energy materials, vitamins, nutrients, antibiotics, etc. If the microbiota fails to ensure the absorption of these substances, even with an excess of essential amino acids in consumed foods, the body will experience a pronounced deficiency of them.
When classifying manifestations of nutritional deficiency, two main conditions are distinguished: kwashiorkor, primarily caused by a protein-deficient diet, and marasmus, a state associated with general protein-caloric malnutrition. Kwashiorkor typically occurs in children aged 1 to 3 years and is characterized by edema, fat retention (especially in the liver, which becomes significantly enlarged), loose skin, sparse hair with no pigment, apathy, and irritability. Marasmus, on the other hand, results in severe growth retardation as early as 6 to 12 months of age. Unlike kwashiorkor-affected children, who often appear bloated and round-faced due to edema, children with marasmus appear withered, with muscle atrophy and no body fat. In classic cases of marasmus, liver, skin, and hair damage are absent: such a child is restless and reactive but not irritable. Between these two extreme conditions, there are many intermediate forms. Practically, it is difficult to distinguish between them, but from a treatment perspective, it is important to determine whether the child’s diet lacked protein, calories, or both.
The underlying principle connecting all these phenomena is based on the idea of energy equivalence between different classes of nutrients and tissue components. When the total calorie intake from primary energy sources (carbohydrates and fats) becomes less than the body's energy expenditure, protein breakdown begins—both dietary and endogenous. This leads to general wasting, affecting muscles and other organs, as observed in marasmus. Thus, starvation can be viewed as a state in which the body "consumes itself" to meet its energy needs. Before this severe and often fatal state occurs, a series of metabolic changes take place, depending on prior nutritional conditions.
Let’s examine the processes occurring in a healthy, well-nourished adult when suddenly deprived of food. Blood glucose levels immediately decrease, leading to reduced insulin secretion and increased glucagon secretion by the pancreas, stimulating hepatic glycogenolysis. If food is absent for 24 hours, liver glycogen stores are largely depleted; by the second day, other mechanisms must be activated. Prolonged glucagon secretion stimulates hormone-sensitive lipase, increasing the release of fatty acids, which are oxidized in the liver. Similarly, ACTH, catecholamines, and other hormones, released in response to stress stimuli affecting regulatory centers, activate adipose tissue lipase. The mobilization of fatty acids from fat stores provides energy to peripheral tissues, the liver, and other internal organs. Unlike short-term energy production from glycogen, triglycerides from fat stores can supply adequate ATP for several weeks.
As the body adapts to prolonged starvation (during the first few weeks), the production of ketone bodies by the liver increases sharply, as fatty acid oxidation predominates, while pyruvate and oxaloacetate levels decrease, limiting the flow of metabolites through the citric acid cycle. After a few weeks, organs such as the heart and, eventually, the brain adjust to meeting a significant portion of their energy needs via ketone bodies. However, if ketone body production exceeds their utilization, metabolic acidosis occurs, which the body attempts to compensate through respiratory CO₂ elimination and renal NH₄⁺ excretion. At the same time, as the body transitions to fatty acid mobilization and oxidation, adipose tissue and the liver progressively deplete key fatty acid biosynthesis enzymes, such as acetyl-CoA carboxylase, fatty acid synthase, and citrate cleavage enzyme.
If starvation persists, tissue proteins are mobilized and used as an energy source. Paradoxically, the first proteins to be broken down are labile proteins of the gastrointestinal tract and digestive enzymes of the pancreas, impairing the utilization of even the small amounts of food that reach the digestive system. Subsequently, proteins from internal organs (liver, spleen) break down, followed by functional muscle proteins, and finally nervous system proteins. This sequence corresponds to normal protein turnover rates in the described metabolic state. Amino acid catabolism, stimulated by elevated glucocorticoid secretion and decreased insulin levels, provides ATP for peripheral tissues and supplies carbon for glucose synthesis in the liver and kidneys.
During the first weeks of starvation, despite an overall increase in protein catabolism, liver enzymes involved in gluconeogenesis, such as aminotransferases, phosphatases, and enzymes catalyzing pyruvate-to-phosphoenolpyruvate conversion, increase significantly. In prolonged starvation, when the brain primarily relies on beta-hydroxybutyrate rather than glucose for energy, gluconeogenesis intensity gradually decreases. Concurrently, muscle mass, overall activity, and energy expenditure decline.
Ultimately, when fat reserves are depleted, the body begins to consume even vital proteins from the heart, lungs, and blood cells, leading to death from circulatory collapse.
GLUCOCORTICOIDS
In response to stimuli such as infection, psycho-emotional stress, trauma, or food intake, the body produces various active substances, including hormones and enzymes.
Glucocorticoids are hormones produced by the body that perform anti-inflammatory functions.
Normally, the body adequately increases the synthesis of glucocorticoids and converts them from an inactive state to an active one. The fluctuation in their release in response to stimuli ranges from 1/50 to 1/200 of the daily dose.
What factors influence the levels and release of glucocorticoids?
Cholesterol is essential for the synthesis of glucocorticoids. Its blood levels are determined by the ratio of high-density and low-density lipoproteins. An insufficient amount of low-density lipoproteins can lead to decreased cholesterol levels in the blood, potentially resulting in inadequate glucocorticoid synthesis.
On the other hand, glucocorticoids may be produced in sufficient amounts but remain in an inactive, bound state.
Using resonant infrared (IR) emitters, it is possible to normalize their concentration. For this purpose, a specialized emitter, GI/ZB, has been developed.
Exposure to this emitter on the gallbladder and bile ducts for one minute is said to reduce inflammation, improve bile duct patency, dissociate lipoprotein complexes, and release free cholesterol.
Applying the emitter to the adrenal glands for the same duration is claimed to convert glucocorticoids from an inactive to an active (free) state and stimulate their synthesis from free cholesterol. These operations are repeated 3-5 times, with a total exposure time of 6-10 minutes. This procedure can be repeated every two hours throughout the day.
These treatments are claimed to help balance high- and low-density lipoprotein levels and dissolve gallstones. They must be performed in a strict sequence without interruption.
Glucocorticoids are hormones produced by the body that perform anti-inflammatory functions.
Normally, the body adequately increases the synthesis of glucocorticoids and converts them from an inactive state to an active one. The fluctuation in their release in response to stimuli ranges from 1/50 to 1/200 of the daily dose.
What factors influence the levels and release of glucocorticoids?
Cholesterol is essential for the synthesis of glucocorticoids. Its blood levels are determined by the ratio of high-density and low-density lipoproteins. An insufficient amount of low-density lipoproteins can lead to decreased cholesterol levels in the blood, potentially resulting in inadequate glucocorticoid synthesis.
On the other hand, glucocorticoids may be produced in sufficient amounts but remain in an inactive, bound state.
Using resonant infrared (IR) emitters, it is possible to normalize their concentration. For this purpose, a specialized emitter, GI/ZB, has been developed.
Exposure to this emitter on the gallbladder and bile ducts for one minute is said to reduce inflammation, improve bile duct patency, dissociate lipoprotein complexes, and release free cholesterol.
Applying the emitter to the adrenal glands for the same duration is claimed to convert glucocorticoids from an inactive to an active (free) state and stimulate their synthesis from free cholesterol. These operations are repeated 3-5 times, with a total exposure time of 6-10 minutes. This procedure can be repeated every two hours throughout the day.
These treatments are claimed to help balance high- and low-density lipoprotein levels and dissolve gallstones. They must be performed in a strict sequence without interruption.
(Note: The medical claims in this text should be critically evaluated, as there is no widely accepted scientific evidence confirming the described effects of infrared emitters on hormone regulation or cholesterol metabolism.)
CHOLESTEROL METABOLISM
Cholesterol (CH) holds a special place among biological compounds. It is a component of tissues, cells, and cell membranes, regulating their permeability, and serves as a precursor for steroid hormones and bile acids. CH is an alcohol capable of forming esters with acetic, benzoic, stearic, and other acids. Its intermediate metabolism occurs in the liver, where it is excreted in its pure form with bile. Cholesterol biosynthesis takes place in all organs and tissues, but bile plays a primary role in this process.
CH enters the gastrointestinal tract through two pathways: from food and as part of intestinal secretions and bile. The absorbed portion of cholesterol, known as the absorption coefficient, is an important physiological indicator. CH is excreted through the intestines, where it is converted into coprostanol. The cholesterol that enters the body is transported to the lymphatic system as part of chylomicrons and very-low-density lipoproteins (VLDL). From there, it reaches the plasma, where it is found in lipoproteins classified into four groups based on density:
- Very-low-density lipoproteins (VLDL)
- Low-density lipoproteins (LDL)
- High-density lipoproteins (HDL)
- Very-high-density lipoproteins (VHDL)
VLDL transport triglycerides from the intestines into the bloodstream, LDL participates in delivering cholesterol into the blood, and HDL removes cholesterol from the body. The liver is the primary supplier of cholesterol to the blood, as it is where cholesterol synthesis occurs. The liver also serves as the first barrier for VLDL and intestinal chylomicrons. What matters most is not the absolute concentration of cholesterol in plasma (which averages 1.9–2.1 g/L) but the ratio of cholesterol in LDL and HDL, as these lipoproteins transport cholesterol through blood vessel walls. Therefore, an imbalance in these lipoproteins can lead to various diseases.
Another issue associated with cholesterol is gallstone disease. Excess cholesterol saturation in bile inevitably leads to the formation of gallstones (primarily cholesterol stones) in the gallbladder and its ducts. In this case, the total cholesterol content in bile is not as important as its phase composition.
Bile is a lipid complex that includes phospholipids, cholesterol, bile acids, and, in adults, cholic acid.
Under normal conditions, cholesterol is kept dissolved in bile by phospholipids and bile acids. However, when liver function is impaired, the composition of bile components changes, leading to the formation of inclusions such as fat droplets, cholesterol esters, and the crystallization of cholesterol. Swelling of lipids can result in the formation of liquid crystals. If bile remains in this state for an extended period, cholesterol can crystallize, meaning that bile stagnation also contributes to gallstone formation.
Hypercholesterolemia (cholesterol levels above 2.6 g/L) is often associated with atherosclerosis and cardiovascular disease.
However, a decrease in plasma cholesterol levels (below 1.5 g/L) can lead to conditions such as hypothyroidism, Addison’s disease, cachexia, and nervous system asthenia. Hypocholesterolemia can also occur in liver diseases such as cirrhosis and infectious hepatitis. Hypercholesterolemia, on the other hand, can be primary (familial, linked to genetic factors) or secondary, caused by external conditions such as obesity, overeating, and physical inactivity.
Another issue associated with cholesterol is gallstone disease. Excess cholesterol saturation in bile inevitably leads to the formation of gallstones (primarily cholesterol stones) in the gallbladder and its ducts. In this case, the total cholesterol content in bile is not as important as its phase composition.
Bile is a lipid complex that includes phospholipids, cholesterol, bile acids, and, in adults, cholic acid.
Under normal conditions, cholesterol is kept dissolved in bile by phospholipids and bile acids. However, when liver function is impaired, the composition of bile components changes, leading to the formation of inclusions such as fat droplets, cholesterol esters, and the crystallization of cholesterol. Swelling of lipids can result in the formation of liquid crystals. If bile remains in this state for an extended period, cholesterol can crystallize, meaning that bile stagnation also contributes to gallstone formation.
Hypercholesterolemia (cholesterol levels above 2.6 g/L) is often associated with atherosclerosis and cardiovascular disease.
However, a decrease in plasma cholesterol levels (below 1.5 g/L) can lead to conditions such as hypothyroidism, Addison’s disease, cachexia, and nervous system asthenia. Hypocholesterolemia can also occur in liver diseases such as cirrhosis and infectious hepatitis. Hypercholesterolemia, on the other hand, can be primary (familial, linked to genetic factors) or secondary, caused by external conditions such as obesity, overeating, and physical inactivity.
The role of cholesterol in LDL and HDL is often linked to their size. It is commonly believed that LDL increases plasma cholesterol concentration because its molecules are larger than those of HDL. However, they can actually be the same size. In our view, the key difference lies in cholesterol saturation: LDL contains significantly more cholesterol, while HDL contains less. According to the laws of solubility, cholesterol will be released into the environment (blood) when its concentration exceeds that of the surrounding medium. Conversely, cholesterol will move into HDL because it is not saturated with cholesterol.
THE CONCEPT OF ACUTE AND CHRONIC PROCESSES
Any chemical processes occurring in the body involve the transfer of electrons, which allows us to speak of the presence of an electric charge on the surface of any organ, its part, or a cell. In other words, all chemical processes have an electrical nature.
Schematically, this can be represented as follows: if we hypothetically define the electrical structure of an organ, each of its points has a specific charge (potential) that ensures the normal metabolic and other functions of the organ. At the same time, the other side of the membrane has the same or a closely matched (coordinated) potential.
The development of a pathological process is accompanied by a change in potential levels. If surrounding organs and tissues, whether tightly or loosely connected, are not involved in the pathological process, the charge on their surface does not change. In other words, processes in other organs remain largely unaffected. A pathological process in which functional changes occur only in a single organ can be considered acute.
In such cases, recovery occurs within a short period.
During disease progression, a potential shift may also occur on the opposite side of the membrane (or even on the membranes of other organs functionally connected to the affected organ). This leads to the involvement of additional organs in the pathological process, altering their electrical potentials as well. In other words, metabolic processes in these organs also change significantly. A pathological process that involves multiple interconnected organs and alters their normal function is considered chronic.
In this case, the connection between organs may be not only anatomical but also functional.
Treatment in such cases requires a longer duration with shorter exposure times to therapeutic radiation and must involve repeated, sequential treatment of all organs included in the pathological process, rather than focusing only on the organ showing clinical manifestations of the disease.
Acute Process:
The functions of the affected organ are altered, but the functions of related organs remain intact. In this case, restoring the function of the affected organ is sufficient to eliminate the disease. The normal function of other organs aids the recovery process.
Chronic Process:
The functions of the affected organ are altered, and the functions of surrounding organs are also compromised. Restoring the affected organ alone does not restore the function of other organs. The altered functions of these organs contribute to disease recurrence. In such cases, a complete restoration of all functionally interconnected organs is necessary.
For a clearer understanding of chronic processes, the following model is useful:
Schematically, this can be represented as follows: if we hypothetically define the electrical structure of an organ, each of its points has a specific charge (potential) that ensures the normal metabolic and other functions of the organ. At the same time, the other side of the membrane has the same or a closely matched (coordinated) potential.
The development of a pathological process is accompanied by a change in potential levels. If surrounding organs and tissues, whether tightly or loosely connected, are not involved in the pathological process, the charge on their surface does not change. In other words, processes in other organs remain largely unaffected. A pathological process in which functional changes occur only in a single organ can be considered acute.
In such cases, recovery occurs within a short period.
During disease progression, a potential shift may also occur on the opposite side of the membrane (or even on the membranes of other organs functionally connected to the affected organ). This leads to the involvement of additional organs in the pathological process, altering their electrical potentials as well. In other words, metabolic processes in these organs also change significantly. A pathological process that involves multiple interconnected organs and alters their normal function is considered chronic.
In this case, the connection between organs may be not only anatomical but also functional.
Treatment in such cases requires a longer duration with shorter exposure times to therapeutic radiation and must involve repeated, sequential treatment of all organs included in the pathological process, rather than focusing only on the organ showing clinical manifestations of the disease.
Acute Process:
The functions of the affected organ are altered, but the functions of related organs remain intact. In this case, restoring the function of the affected organ is sufficient to eliminate the disease. The normal function of other organs aids the recovery process.
Chronic Process:
The functions of the affected organ are altered, and the functions of surrounding organs are also compromised. Restoring the affected organ alone does not restore the function of other organs. The altered functions of these organs contribute to disease recurrence. In such cases, a complete restoration of all functionally interconnected organs is necessary.
For a clearer understanding of chronic processes, the following model is useful:
Imagine replacing a diseased organ (whose altered potentials have affected functionally connected organs) with a healthy one (with normal potentials). Under the influence of the altered potentials of the connected organs, the healthy organ’s potential will also change, disrupting its function.
PHASES OF THE COURSE OF A PATHOLOGICAL PRECESS
In the development of any pathological process, three main phases are distinguished.
In the development of any pathological process, three main phases are distinguished.
In this phase, reversible processes occur (i.e., their normalization requires only minimal external intervention).
During Voll testing, most patients show readings above 75. The body's processes are compensated, all organs and tissues are coping with the load, and they strive to bring the body into a state of dynamic equilibrium.
Example: A patient undergoing Voll testing shows below-normal readings at the control point of the cortical layer of the kidneys. The patient does not report any pain in the kidney area, and the urine analysis is normal. Emitters GI, AF, and RV do not test at the cortical layer kidney point, indicating the absence of a pathological process in this organ. However, the emitter ZB proves effective when applied to arterial points, improving readings at kidney cortical layer points. This suggests that the primary pathological process is localized in the blood vessels, meaning that although the kidneys are not yet involved in the pathological process, they struggle to handle the workload due to impaired blood flow. If this condition progresses, an inflammatory process may develop in the kidneys, and GI series emitters will begin to test at kidney points.
Phase transition: from point 1 to point 2
In this phase, one or two organs show below-normal EAV (Electroacupuncture according to Voll) readings, while other points remain within the normal range. Readings above the normal range may indicate a compensatory increase in functional activity caused by the presence of a pathological process in the organ with low EAV readings.
Example: A patient complains of frequent headaches. EAV readings at vascular points are above normal, while readings at the large intestine point are low. When testing emitters at the large intestine points, normalization of readings was observed after exposure to the GI emitter on the large intestine area, indicating the presence of an inflammatory process. Simultaneously, vascular point readings also normalized. After several sessions of infrared GI emitter therapy on the intestine, EAV readings at the large intestine points increased, vascular readings normalized, and the patient's headaches significantly decreased.
Phase transition: below point 2
Patients report pain in various organs. EAV testing shows an increasing number of points with below-normal readings, indicating that more organs are becoming involved in the pathological process. In the early stages, a decrease in the bioelectric potential of organs may not be due to a pathological process but rather to insufficient compensatory capacity.
During Voll testing, most patients show readings above 75. The body's processes are compensated, all organs and tissues are coping with the load, and they strive to bring the body into a state of dynamic equilibrium.
Example: A patient undergoing Voll testing shows below-normal readings at the control point of the cortical layer of the kidneys. The patient does not report any pain in the kidney area, and the urine analysis is normal. Emitters GI, AF, and RV do not test at the cortical layer kidney point, indicating the absence of a pathological process in this organ. However, the emitter ZB proves effective when applied to arterial points, improving readings at kidney cortical layer points. This suggests that the primary pathological process is localized in the blood vessels, meaning that although the kidneys are not yet involved in the pathological process, they struggle to handle the workload due to impaired blood flow. If this condition progresses, an inflammatory process may develop in the kidneys, and GI series emitters will begin to test at kidney points.
Phase transition: from point 1 to point 2
In this phase, one or two organs show below-normal EAV (Electroacupuncture according to Voll) readings, while other points remain within the normal range. Readings above the normal range may indicate a compensatory increase in functional activity caused by the presence of a pathological process in the organ with low EAV readings.
Example: A patient complains of frequent headaches. EAV readings at vascular points are above normal, while readings at the large intestine point are low. When testing emitters at the large intestine points, normalization of readings was observed after exposure to the GI emitter on the large intestine area, indicating the presence of an inflammatory process. Simultaneously, vascular point readings also normalized. After several sessions of infrared GI emitter therapy on the intestine, EAV readings at the large intestine points increased, vascular readings normalized, and the patient's headaches significantly decreased.
Phase transition: below point 2
Patients report pain in various organs. EAV testing shows an increasing number of points with below-normal readings, indicating that more organs are becoming involved in the pathological process. In the early stages, a decrease in the bioelectric potential of organs may not be due to a pathological process but rather to insufficient compensatory capacity.
GENERAL PRINCIPLES OF OPERATION AND SELECTION OF INFRARED (IR) EMITTERS
Selection of the emitter depends on the diagnosis established after examination, as well as on the nosodes of toxins, infections, and organ functions identified through EAF (Electroacupuncture by Voll). Its type can also be determined during EAF diagnostics.
To correctly choose an IR emitter, it must be placed in the projection of the affected organ while simultaneously measuring the bioelectric potential of the corresponding point using the Voll device. This point reflects the state of the organ or its part, the control point on the SPED meridian, or a point on the SPED meridian corresponding to the affected level.
If the initial EAF readings are above or below normal, the correctly selected emitter will cause the values to decrease or increase, respectively, until they reach normal levels. If, after 15-20 seconds of exposure in the affected area, the readings on the Voll device do not change or deviate further from the norm, the emitter is incorrectly chosen.
The exposure time of the emitters depends on the patient's condition, the body's resistance and adaptive capabilities, the extent of the pathological process (its stage and severity), and can be determined using EAF.
The sequence of applying the lamps in the treatment scheme is determined by the type, stage, and phase of the pathological process.
General and localized types of emitters can be used simultaneously (under the control of the Voll device).
Treatment should begin after:
To correctly choose an IR emitter, it must be placed in the projection of the affected organ while simultaneously measuring the bioelectric potential of the corresponding point using the Voll device. This point reflects the state of the organ or its part, the control point on the SPED meridian, or a point on the SPED meridian corresponding to the affected level.
If the initial EAF readings are above or below normal, the correctly selected emitter will cause the values to decrease or increase, respectively, until they reach normal levels. If, after 15-20 seconds of exposure in the affected area, the readings on the Voll device do not change or deviate further from the norm, the emitter is incorrectly chosen.
The exposure time of the emitters depends on the patient's condition, the body's resistance and adaptive capabilities, the extent of the pathological process (its stage and severity), and can be determined using EAF.
The sequence of applying the lamps in the treatment scheme is determined by the type, stage, and phase of the pathological process.
General and localized types of emitters can be used simultaneously (under the control of the Voll device).
Treatment should begin after:
- Balancing the sympathetic and parasympathetic divisions of the autonomic nervous system (by using KL/KH emitters on the hypothalamus), determining the body's resistance state and its adaptive capabilities;
- Normalizing intestinal function;
- Normalizing lipid metabolism;
- Normalizing adrenal gland activity.
In the presence of infections, inflammatory processes, circulatory disorders, etc., depending on the patient's condition, a general course of therapy is prescribed:
Note: The general course may last 1-3 weeks. The exposure time for ZB in the second week is 10 minutes, and in the third week, it is 15 minutes. The exposure time increases gradually to prevent a sharp intoxication of the body.
COMBINATION OF INFRARED THERAPY WITH MEDICATION THERAPY
Due to habits acquired through years of experience, doctors often feel inclined to prescribe antibiotics to alleviate a patient's condition. In the absence of resonance infrared (IR) emitters, such actions are justified, as they can often provide real relief and, in many cases, even save a patient's life. However, when treating with resonance IR emitters, the use of antibiotics is unnecessary and may even lead to certain harmful consequences.
The reason lies in the fact that the normal intestinal microflora supplies essential plastic and energy-related materials, vitamins, nutrients, and even natural antibiotics needed for proper metabolism. The use of antibiotics completely alters the composition of this microflora. The gut microbiome is also referred to as the "cradle of immunity," and any disruption in its balance drastically reduces the body's immune defense and ability to resist external and internal destabilizing factors. Therefore, it is advisable to avoid antibiotics when using IR therapy.
A study published in New Scientist on June 12, 1999, reported that a team of researchers led by Dr. Lynda Chin from Dana-Farber Cancer Institute developed a strain of mice with a biological "switch" that could turn melanoma development on and off.
When these mice received a widely used antibiotic in their drinking water, it activated a cancer-causing gene, leading to melanoma tumor growth within two to three months. When the antibiotic was discontinued, the process was reversed, and the tumors shrank.
Antibiotics such as chloramphenicol (levomycetin), actinomycin D, and hormones like methyltestosterone, progesterone, and prednisone have often been linked to the development of malignant tumors.
The same applies to the use of hormonal drugs. Throughout the practice of IR therapy, there has not been a single case where treatment could not be conducted without hormonal medications. Moreover, patients who had been taking hormonal drugs before undergoing IR therapy gradually discontinued them as they recovered, as these medications became unnecessary.
The reason lies in the fact that the normal intestinal microflora supplies essential plastic and energy-related materials, vitamins, nutrients, and even natural antibiotics needed for proper metabolism. The use of antibiotics completely alters the composition of this microflora. The gut microbiome is also referred to as the "cradle of immunity," and any disruption in its balance drastically reduces the body's immune defense and ability to resist external and internal destabilizing factors. Therefore, it is advisable to avoid antibiotics when using IR therapy.
A study published in New Scientist on June 12, 1999, reported that a team of researchers led by Dr. Lynda Chin from Dana-Farber Cancer Institute developed a strain of mice with a biological "switch" that could turn melanoma development on and off.
When these mice received a widely used antibiotic in their drinking water, it activated a cancer-causing gene, leading to melanoma tumor growth within two to three months. When the antibiotic was discontinued, the process was reversed, and the tumors shrank.
Antibiotics such as chloramphenicol (levomycetin), actinomycin D, and hormones like methyltestosterone, progesterone, and prednisone have often been linked to the development of malignant tumors.
The same applies to the use of hormonal drugs. Throughout the practice of IR therapy, there has not been a single case where treatment could not be conducted without hormonal medications. Moreover, patients who had been taking hormonal drugs before undergoing IR therapy gradually discontinued them as they recovered, as these medications became unnecessary.
CONCEPT OF DRAINAGE AND ITS POSSIBLE MANIFESTATIONS
Drainage is the process of eliminating all types of toxins from the body. Physiologically, it occurs through urine, feces, sweat, and the respiratory tract. In women, drainage also takes place during the menstrual cycle. During therapeutic procedures, a large amount of toxins enters the bloodstream, including microbial and viral breakdown products, connective tissue remnants, tumor fragments, atherosclerotic deposits in blood vessels, etc.
A normal protective reaction of the body is to accelerate the removal of foreign substances. As a result, cardiac output increases, leading to elevated blood pressure and a faster pulse. If the body's adaptive capacity is high, it can handle this load, and the toxins released due to infrared (IR) exposure are easily eliminated from the bloodstream without affecting the patient's overall condition. However, if the body's resistance is low and it struggles to eliminate toxins, intoxication symptoms may worsen. These symptoms include increased weakness, drowsiness, headaches, nausea, vomiting, diarrhea, skin rashes, increased urination, and possible exacerbation of existing or past illnesses. If the body's adaptive capabilities are sufficient, these symptoms are usually mild and do not require adjustments to IR therapy.
To enhance resistance, appropriate IR emitters are prescribed. To reduce intoxication, patients are advised to drink plenty of fluids (green tea, juices, water), consumed slowly in small sips (one glass over 15 minutes). On average, 1-2 liters per day is sufficient, with fluid intake reduced or stopped two hours before sleep. If intoxication symptoms persist, intravenous detoxification solutions such as Ringer's solution, Hemodez, Reopoliglyukin, Acesol, or Dissol may be administered.
In cases of severe intoxication, in addition to standard measures, a rapid and effective way to alleviate symptoms is the simultaneous use of RC+ZB emitters. The RC emitter significantly reduces free radical levels, while the ZB emitter facilitates the rapid removal of metabolic waste. A 10-minute exposure is usually sufficient to relieve intoxication symptoms, and this approach is also effective for other, non-treatment-related intoxications.
Patients with low adaptive capacity may experience pronounced clinical symptoms due to even minor homeostatic changes. If drainage primarily occurs through the kidneys, patients may notice cloudy urine and increased urination. Close monitoring of urine tests is necessary to prevent complications such as the appearance of red blood cells, casts, or protein in the urine.
If the vascular system is affected, changes in pulse or blood pressure may occur. In hypertensive patients, blood pressure may rise. Individuals with cardiac rhythm disorders or tachycardia may experience significant worsening of their condition even with minor pulse increases, leading to arrhythmias. In such cases, treatment duration should be reduced, and the functional state of the adrenal glands, pancreas, and autonomic nervous system must be carefully monitored. The exposure time and emitter type should be determined based on electroacupuncture (EAF) testing and continuous monitoring of pulse and blood pressure.
Some patients may develop a fever due to increased anaerobic metabolism resulting from oxygen deficiency. Anaerobic metabolism is significantly less efficient than aerobic metabolism, producing only 2 ATP molecules per glucose molecule compared to 38 ATP molecules via aerobic metabolism. If the fever does not exceed 38°C, fluid intake should be increased, and an enema may be performed. If the fever rises above 38°C but the patient tolerates it well, antipyretic medications should not be used. These drugs are only administered in cases of severe illness.
Skin manifestations can include various rashes (vesicles, papules, pustules, urticaria). Individuals with pre-existing skin conditions may experience a worsening of their symptoms due to predominant drainage through the skin.
Joint pain may intensify due to the release of toxins into the bloodstream, impairing blood supply to the joints.
It is nearly impossible to describe all possible drainage manifestations, as each patient's response is highly individual. If any symptoms indicating a worsening condition appear, it is necessary to measure indicators at adrenal, autonomic nervous system, pancreas, and metabolic regulation points.
As mentioned earlier, one of the liver's primary functions is detoxification. The liver is connected to the intestines only through the gallbladder.
This suggests that effective drainage is only possible when the gallbladder has adequate capacity. The function of the small and large intestines must also be normal. For example, in cases of constipation, the drainage system via the intestines functions poorly. Additionally, the liver cannot supply the brain with sufficient glucose, which is another of its key functions. As a result, the kidneys increase glucose synthesis, leading to dysfunction.
Over the past years, sugar consumption has increased sharply. Sugar is rapidly absorbed and converted into fat, forming low-density lipoproteins. This raises cholesterol levels in the blood, disrupts microcirculation, and reduces the gallbladder's capacity. Consequently, liver and kidney excretory functions become impaired, leading to increased waste concentration in the blood and reduced oxygen levels. As a result, the heart and brain suffer from oxygen deficiency, increasing the likelihood of oxygen utilization in the form of superoxide. This significantly raises the risk of cardiovascular and oncological diseases.
A century ago, physical labor was more demanding. Without tractors and machinery, energy expenditure during tasks such as mowing reached 7,000 kcal per day, while food intake replenished only about 5,000 kcal. To prevent exhaustion, high-energy foods rich in simple carbohydrates were essential to quickly restore energy balance.
The relationship between lifestyle and overall health will be discussed in future publications.
A normal protective reaction of the body is to accelerate the removal of foreign substances. As a result, cardiac output increases, leading to elevated blood pressure and a faster pulse. If the body's adaptive capacity is high, it can handle this load, and the toxins released due to infrared (IR) exposure are easily eliminated from the bloodstream without affecting the patient's overall condition. However, if the body's resistance is low and it struggles to eliminate toxins, intoxication symptoms may worsen. These symptoms include increased weakness, drowsiness, headaches, nausea, vomiting, diarrhea, skin rashes, increased urination, and possible exacerbation of existing or past illnesses. If the body's adaptive capabilities are sufficient, these symptoms are usually mild and do not require adjustments to IR therapy.
To enhance resistance, appropriate IR emitters are prescribed. To reduce intoxication, patients are advised to drink plenty of fluids (green tea, juices, water), consumed slowly in small sips (one glass over 15 minutes). On average, 1-2 liters per day is sufficient, with fluid intake reduced or stopped two hours before sleep. If intoxication symptoms persist, intravenous detoxification solutions such as Ringer's solution, Hemodez, Reopoliglyukin, Acesol, or Dissol may be administered.
In cases of severe intoxication, in addition to standard measures, a rapid and effective way to alleviate symptoms is the simultaneous use of RC+ZB emitters. The RC emitter significantly reduces free radical levels, while the ZB emitter facilitates the rapid removal of metabolic waste. A 10-minute exposure is usually sufficient to relieve intoxication symptoms, and this approach is also effective for other, non-treatment-related intoxications.
Patients with low adaptive capacity may experience pronounced clinical symptoms due to even minor homeostatic changes. If drainage primarily occurs through the kidneys, patients may notice cloudy urine and increased urination. Close monitoring of urine tests is necessary to prevent complications such as the appearance of red blood cells, casts, or protein in the urine.
If the vascular system is affected, changes in pulse or blood pressure may occur. In hypertensive patients, blood pressure may rise. Individuals with cardiac rhythm disorders or tachycardia may experience significant worsening of their condition even with minor pulse increases, leading to arrhythmias. In such cases, treatment duration should be reduced, and the functional state of the adrenal glands, pancreas, and autonomic nervous system must be carefully monitored. The exposure time and emitter type should be determined based on electroacupuncture (EAF) testing and continuous monitoring of pulse and blood pressure.
Some patients may develop a fever due to increased anaerobic metabolism resulting from oxygen deficiency. Anaerobic metabolism is significantly less efficient than aerobic metabolism, producing only 2 ATP molecules per glucose molecule compared to 38 ATP molecules via aerobic metabolism. If the fever does not exceed 38°C, fluid intake should be increased, and an enema may be performed. If the fever rises above 38°C but the patient tolerates it well, antipyretic medications should not be used. These drugs are only administered in cases of severe illness.
Skin manifestations can include various rashes (vesicles, papules, pustules, urticaria). Individuals with pre-existing skin conditions may experience a worsening of their symptoms due to predominant drainage through the skin.
Joint pain may intensify due to the release of toxins into the bloodstream, impairing blood supply to the joints.
It is nearly impossible to describe all possible drainage manifestations, as each patient's response is highly individual. If any symptoms indicating a worsening condition appear, it is necessary to measure indicators at adrenal, autonomic nervous system, pancreas, and metabolic regulation points.
As mentioned earlier, one of the liver's primary functions is detoxification. The liver is connected to the intestines only through the gallbladder.
This suggests that effective drainage is only possible when the gallbladder has adequate capacity. The function of the small and large intestines must also be normal. For example, in cases of constipation, the drainage system via the intestines functions poorly. Additionally, the liver cannot supply the brain with sufficient glucose, which is another of its key functions. As a result, the kidneys increase glucose synthesis, leading to dysfunction.
Over the past years, sugar consumption has increased sharply. Sugar is rapidly absorbed and converted into fat, forming low-density lipoproteins. This raises cholesterol levels in the blood, disrupts microcirculation, and reduces the gallbladder's capacity. Consequently, liver and kidney excretory functions become impaired, leading to increased waste concentration in the blood and reduced oxygen levels. As a result, the heart and brain suffer from oxygen deficiency, increasing the likelihood of oxygen utilization in the form of superoxide. This significantly raises the risk of cardiovascular and oncological diseases.
A century ago, physical labor was more demanding. Without tractors and machinery, energy expenditure during tasks such as mowing reached 7,000 kcal per day, while food intake replenished only about 5,000 kcal. To prevent exhaustion, high-energy foods rich in simple carbohydrates were essential to quickly restore energy balance.
The relationship between lifestyle and overall health will be discussed in future publications.
INSTALLATION "ST"
The development of effective methods for treating diseases of the pelvic organs, as well as the body as a whole, is a pressing issue among clinicians. This problem becomes even more significant considering the increasing number of individuals with diseases of the reproductive organs, intestines, and other systems, along with a growing prevalence of allergic reactions to medications. We cannot blame pharmacists for this, as we ourselves disrupt the natural metabolic processes initially embedded in our bodies.
Based on the method of resonance therapy, a fundamentally new installation, "ST", has been developed, successfully applied for the treatment of various diseases of the pelvic organs and intestines.
The "ST" installation consists of GI and KL emitters. These emitters act directly on the lungs, perineum, and pelvic organs.
Using the "ST" installation, significant therapeutic effects can be achieved in the treatment of colpitis, vulvovaginitis, proctitis, paraproctitis, endometritis, adnexitis, prostatitis, cystitis, urethritis, prostate adenoma, fibroids, malignant and benign tumors of the reproductive organs, cervical erosion, hemorrhoids, rectal fissures, bronchitis, and more—all without the use of medication.
By acting on pathological processes through the anterior abdominal wall and perineum, the time required for the disappearance of clinical symptoms is significantly reduced.
The combination of general and local emitters with the "ST" installation allows for rapid achievement of positive treatment results.
A comparative analysis of results has shown that the effectiveness of treatment with the "ST" installation depends not only on the combination of local and general emitters and their exposure time but also on the individual susceptibility of the body's immune system. A standardized approach to treating each patient cannot be applied; the entire therapy period must be monitored using EAF diagnostics (to determine exposure time, emitters, and their combination).
It has been established that using this installation normalizes the immune system.
Under the influence of infrared (IR) radiation on the rectum, sigmoid colon, bladder, reproductive organs, and glands, in addition to reducing inflammation, eliminating viruses, and improving blood supply, there is also an increased production of secretory IgA. This immunoglobulin is produced in the mucous membranes of the urogenital organs and the gastrointestinal tract. It is known that a decrease in secretory IgA levels leads to an increase in pathogenic microflora in the intestines and reproductive organs. IgA ensures local immunity of the mucous membranes, including the female reproductive organs, and has antiviral and antibacterial effects.
Simultaneous exposure of the KL(s) emitter to the thymus gland promotes the production of IgM (γ-macroglobulin), which is synthesized with the participation of informational RNA on ribosomes of the granular endoplasmic reticulum of lymphocytes and plasmocytes. IgM has antiviral and bacteriological activity, which is crucial for our body's defensive response to foreign agents entering through so-called "gateway" sites.
Based on the method of resonance therapy, a fundamentally new installation, "ST", has been developed, successfully applied for the treatment of various diseases of the pelvic organs and intestines.
The "ST" installation consists of GI and KL emitters. These emitters act directly on the lungs, perineum, and pelvic organs.
Using the "ST" installation, significant therapeutic effects can be achieved in the treatment of colpitis, vulvovaginitis, proctitis, paraproctitis, endometritis, adnexitis, prostatitis, cystitis, urethritis, prostate adenoma, fibroids, malignant and benign tumors of the reproductive organs, cervical erosion, hemorrhoids, rectal fissures, bronchitis, and more—all without the use of medication.
By acting on pathological processes through the anterior abdominal wall and perineum, the time required for the disappearance of clinical symptoms is significantly reduced.
The combination of general and local emitters with the "ST" installation allows for rapid achievement of positive treatment results.
A comparative analysis of results has shown that the effectiveness of treatment with the "ST" installation depends not only on the combination of local and general emitters and their exposure time but also on the individual susceptibility of the body's immune system. A standardized approach to treating each patient cannot be applied; the entire therapy period must be monitored using EAF diagnostics (to determine exposure time, emitters, and their combination).
It has been established that using this installation normalizes the immune system.
Under the influence of infrared (IR) radiation on the rectum, sigmoid colon, bladder, reproductive organs, and glands, in addition to reducing inflammation, eliminating viruses, and improving blood supply, there is also an increased production of secretory IgA. This immunoglobulin is produced in the mucous membranes of the urogenital organs and the gastrointestinal tract. It is known that a decrease in secretory IgA levels leads to an increase in pathogenic microflora in the intestines and reproductive organs. IgA ensures local immunity of the mucous membranes, including the female reproductive organs, and has antiviral and antibacterial effects.
Simultaneous exposure of the KL(s) emitter to the thymus gland promotes the production of IgM (γ-macroglobulin), which is synthesized with the participation of informational RNA on ribosomes of the granular endoplasmic reticulum of lymphocytes and plasmocytes. IgM has antiviral and bacteriological activity, which is crucial for our body's defensive response to foreign agents entering through so-called "gateway" sites.
Let's provide example treatment schemes for some diseases using the "ST" device.
Acute Prostatitis
10-15 minutes, 2 times a day for 8-10 days.
Chronic prostatitis
15 minutes per day for 15 days.
Urethritis
For bacterial etiology: 20-25 minutes for 7 days.
Impotence
For 10-20 minutes over the course of 1 month.
Hemorrhoids
10 minutes, twice a day for 3-4 days.
Enuresis
For 10-15 minutes over 6-7 days. If a virus is detected during EAF diagnostics, an emitter from the R series should be added to the treatment.
Uterine fibroid
For 10-15 minutes, 1-2 times a day for 3-4 weeks.
Ovarian cyst
For 10 minutes over a period of 2-3 weeks.
Acute endometritis
For 15-20 minutes over 7-8 days.
Chronic endometritis
For 10-15 minutes over 2-3 weeks.
Postpartum endometritis
15-20 minutes for a week
Uterine bleeding: 10 minutes for 3-4 days.
Uterine bleeding: 10 minutes for 3-4 days.
Infertility
For 10-15 minutes over 2-3 weeks.
Bartholinitis
for 15-20 minutes over 8-10 days.
Frequent and spontaneous miscarriages (spontaneous abortion)
for 20 minutes over 2-3 weeks.
Diabetes mellitus (glucosuria)
for 10-15 minutes over 10-12 days.
Angiopathy
for 10 minutes over 2 weeks.
Intestinal colic
for 10-15 minutes, 2-3 times a day.
Cholecystitis
for 10 minutes over 8-10 days.
Colitis
For 10-15 minutes over 5-6 days.
General exhaustion, loss of strength, postoperative condition.
For 10-15 minutes over 10-12 days.
Endarteritis
for 10 minutes over 2 weeks
Varicose veins
for 10-15 minutes over 2-3 weeks
lumbar-sacral osteochondrosis
for 10-15 minutes over a period of 2 weeks
Neurasthenia
for 10 minutes over a period of 2 weeks.
Polyneuritis for a period of 2 weeks.
for 15 minutes.